Name | Number of supported studies | Average coverage | |
---|---|---|---|
lung | 18 studies | 39% ± 12% | |
peripheral blood | 16 studies | 35% ± 12% | |
intestine | 13 studies | 39% ± 15% | |
kidney | 9 studies | 43% ± 11% | |
brain | 9 studies | 31% ± 11% | |
eye | 8 studies | 49% ± 19% | |
lymph node | 7 studies | 41% ± 16% | |
liver | 6 studies | 41% ± 21% | |
pancreas | 5 studies | 52% ± 25% | |
bone marrow | 5 studies | 29% ± 7% | |
uterus | 5 studies | 52% ± 7% | |
placenta | 4 studies | 31% ± 11% | |
prostate | 4 studies | 61% ± 19% | |
breast | 4 studies | 54% ± 9% | |
heart | 3 studies | 26% ± 7% | |
adrenal gland | 3 studies | 45% ± 9% | |
esophagus | 3 studies | 54% ± 13% | |
skin | 3 studies | 43% ± 16% | |
thymus | 3 studies | 59% ± 9% |
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
esophagus | 100% | 5663.34 | 1442 / 1445 | 100% | 117.14 | 183 / 183 |
brain | 100% | 8132.79 | 2632 / 2642 | 100% | 90.29 | 705 / 705 |
bladder | 100% | 4643.95 | 21 / 21 | 100% | 90.45 | 502 / 504 |
adrenal gland | 100% | 6367.07 | 258 / 258 | 100% | 90.90 | 229 / 230 |
kidney | 100% | 7682.75 | 89 / 89 | 99% | 83.69 | 896 / 901 |
stomach | 100% | 5442.33 | 358 / 359 | 100% | 115.76 | 285 / 286 |
intestine | 100% | 7856.45 | 966 / 966 | 99% | 128.61 | 523 / 527 |
breast | 99% | 3903.00 | 456 / 459 | 100% | 72.94 | 1113 / 1118 |
thymus | 99% | 4259.18 | 649 / 653 | 99% | 50.26 | 601 / 605 |
liver | 98% | 4725.17 | 222 / 226 | 100% | 58.53 | 405 / 406 |
lung | 98% | 4202.94 | 566 / 578 | 100% | 89.88 | 1155 / 1155 |
skin | 98% | 4247.09 | 1774 / 1809 | 99% | 87.89 | 466 / 472 |
prostate | 97% | 3411.91 | 237 / 245 | 99% | 60.02 | 498 / 502 |
uterus | 94% | 2874.83 | 160 / 170 | 100% | 86.29 | 459 / 459 |
ovary | 87% | 3503.88 | 156 / 180 | 100% | 87.68 | 430 / 430 |
pancreas | 57% | 1138.31 | 186 / 328 | 98% | 56.91 | 175 / 178 |
lymph node | 0% | 0 | 0 / 0 | 100% | 72.25 | 29 / 29 |
muscle | 100% | 14770.27 | 803 / 803 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 100% | 91.08 | 45 / 45 |
ureter | 0% | 0 | 0 / 0 | 100% | 31.01 | 1 / 1 |
spleen | 100% | 3508.32 | 240 / 241 | 0% | 0 | 0 / 0 |
adipose | 99% | 4622.97 | 1197 / 1204 | 0% | 0 | 0 / 0 |
blood vessel | 99% | 4581.00 | 1318 / 1335 | 0% | 0 | 0 / 0 |
heart | 98% | 20658.63 | 841 / 861 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 93% | 47.12 | 74 / 80 |
peripheral blood | 53% | 3716.48 | 489 / 929 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
GO_0097194 | Biological process | execution phase of apoptosis |
GO_0097193 | Biological process | intrinsic apoptotic signaling pathway |
GO_0006122 | Biological process | mitochondrial electron transport, ubiquinol to cytochrome c |
GO_0045333 | Biological process | cellular respiration |
GO_0043280 | Biological process | positive regulation of cysteine-type endopeptidase activity involved in apoptotic process |
GO_0006123 | Biological process | mitochondrial electron transport, cytochrome c to oxygen |
GO_0070469 | Cellular component | respirasome |
GO_0005758 | Cellular component | mitochondrial intermembrane space |
GO_0005829 | Cellular component | cytosol |
GO_0043293 | Cellular component | apoptosome |
GO_0005743 | Cellular component | mitochondrial inner membrane |
GO_0005739 | Cellular component | mitochondrion |
GO_0005634 | Cellular component | nucleus |
GO_0020037 | Molecular function | heme binding |
GO_0005515 | Molecular function | protein binding |
GO_0046872 | Molecular function | metal ion binding |
GO_0009055 | Molecular function | electron transfer activity |
Gene name | CYCS |
Protein name | Cytochrome c |
Synonyms | CYC |
Description | FUNCTION: Electron carrier protein. The oxidized form of the cytochrome c heme group can accept an electron from the heme group of the cytochrome c1 subunit of cytochrome reductase. Cytochrome c then transfers this electron to the cytochrome oxidase complex, the final protein carrier in the mitochondrial electron-transport chain.; FUNCTION: Plays a role in apoptosis. Suppression of the anti-apoptotic members or activation of the pro-apoptotic members of the Bcl-2 family leads to altered mitochondrial membrane permeability resulting in release of cytochrome c into the cytosol. Binding of cytochrome c to Apaf-1 triggers the activation of caspase-9, which then accelerates apoptosis by activating other caspases. FUNCTION: Electron carrier protein. The oxidized form of the cytochrome c heme group can accept an electron from the heme group of the cytochrome c1 subunit of cytochrome reductase. Cytochrome c then transfers this electron to the cytochrome oxidase complex, the final protein carrier in the mitochondrial electron-transport chain. .; FUNCTION: Plays a role in apoptosis. Suppression of the anti-apoptotic members or activation of the pro-apoptotic members of the Bcl-2 family leads to altered mitochondrial membrane permeability resulting in release of cytochrome c into the cytosol. Binding of cytochrome c to Apaf-1 triggers the activation of caspase-9, which then accelerates apoptosis by activating other caspases. . FUNCTION: Electron carrier protein. The oxidized form of the cytochrome c heme group can accept an electron from the heme group of the cytochrome c1 subunit of cytochrome reductase. Cytochrome c then transfers this electron to the cytochrome oxidase complex, the final protein carrier in the mitochondrial electron-transport chain. .; FUNCTION: Plays a role in apoptosis. Suppression of the anti-apoptotic members or activation of the pro-apoptotic members of the Bcl-2 family leads to altered mitochondrial membrane permeability resulting in release of cytochrome c into the cytosol. Binding of cytochrome c to Apaf-1 triggers the activation of caspase-9, which then accelerates apoptosis by activating other caspases. . |
Accessions | P99999 ENST00000409764.5 G4XXL9 ENST00000305786.7 ENST00000409409.5 ENST00000413447.1 C9JFR7 |