Name | Number of supported studies | Average coverage | |
---|---|---|---|
endothelial cell | 38 studies | 35% ± 13% | |
capillary endothelial cell | 13 studies | 30% ± 8% | |
vein endothelial cell | 13 studies | 40% ± 21% | |
endothelial cell of artery | 12 studies | 33% ± 9% | |
oligodendrocyte precursor cell | 7 studies | 35% ± 16% | |
endothelial cell of vascular tree | 6 studies | 35% ± 19% | |
glomerular endothelial cell | 4 studies | 57% ± 4% | |
endothelial cell of lymphatic vessel | 4 studies | 33% ± 8% | |
glutamatergic neuron | 3 studies | 19% ± 3% |
Insufficient scRNA-seq data for expression of TEK at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
breast | 100% | 3283.62 | 459 / 459 | 93% | 9.93 | 1044 / 1118 |
thymus | 100% | 2321.03 | 653 / 653 | 90% | 13.08 | 547 / 605 |
brain | 94% | 910.06 | 2474 / 2642 | 96% | 9.72 | 680 / 705 |
lung | 100% | 5691.83 | 578 / 578 | 84% | 6.28 | 968 / 1155 |
prostate | 90% | 767.08 | 220 / 245 | 91% | 6.62 | 457 / 502 |
kidney | 100% | 2675.98 | 89 / 89 | 80% | 19.19 | 720 / 901 |
adrenal gland | 100% | 1607.91 | 257 / 258 | 77% | 9.02 | 178 / 230 |
intestine | 99% | 1170.69 | 953 / 966 | 75% | 4.59 | 393 / 527 |
stomach | 86% | 674.84 | 309 / 359 | 84% | 5.95 | 239 / 286 |
esophagus | 83% | 932.81 | 1199 / 1445 | 78% | 5.09 | 143 / 183 |
bladder | 90% | 905.29 | 19 / 21 | 66% | 3.62 | 335 / 504 |
uterus | 99% | 1673.99 | 169 / 170 | 54% | 2.81 | 247 / 459 |
pancreas | 40% | 209.62 | 132 / 328 | 95% | 10.24 | 169 / 178 |
skin | 60% | 432.71 | 1091 / 1809 | 53% | 3.34 | 252 / 472 |
ovary | 69% | 569.85 | 125 / 180 | 39% | 1.54 | 169 / 430 |
adipose | 100% | 3902.77 | 1204 / 1204 | 0% | 0 | 0 / 0 |
spleen | 100% | 2531.15 | 241 / 241 | 0% | 0 | 0 / 0 |
heart | 100% | 1900.06 | 859 / 861 | 0% | 0 | 0 / 0 |
liver | 41% | 249.13 | 92 / 226 | 57% | 3.20 | 232 / 406 |
blood vessel | 96% | 1283.01 | 1285 / 1335 | 0% | 0 | 0 / 0 |
muscle | 87% | 594.23 | 699 / 803 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 53% | 2.58 | 24 / 45 |
lymph node | 0% | 0 | 0 / 0 | 48% | 1.52 | 14 / 29 |
eye | 0% | 0 | 0 / 0 | 14% | 0.46 | 11 / 80 |
peripheral blood | 0% | 1.22 | 2 / 929 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 1 |
GO_0001525 | Biological process | angiogenesis |
GO_2000352 | Biological process | negative regulation of endothelial cell apoptotic process |
GO_0007169 | Biological process | cell surface receptor protein tyrosine kinase signaling pathway |
GO_0043434 | Biological process | response to peptide hormone |
GO_0043627 | Biological process | response to estrogen |
GO_0007507 | Biological process | heart development |
GO_0045766 | Biological process | positive regulation of angiogenesis |
GO_0060347 | Biological process | heart trabecula formation |
GO_0043114 | Biological process | regulation of vascular permeability |
GO_0010595 | Biological process | positive regulation of endothelial cell migration |
GO_0007275 | Biological process | multicellular organism development |
GO_1902533 | Biological process | positive regulation of intracellular signal transduction |
GO_0001934 | Biological process | positive regulation of protein phosphorylation |
GO_0001958 | Biological process | endochondral ossification |
GO_0043410 | Biological process | positive regulation of MAPK cascade |
GO_0035022 | Biological process | positive regulation of Rac protein signal transduction |
GO_0007267 | Biological process | cell-cell signaling |
GO_0002040 | Biological process | sprouting angiogenesis |
GO_0016525 | Biological process | negative regulation of angiogenesis |
GO_0033674 | Biological process | positive regulation of kinase activity |
GO_2000351 | Biological process | regulation of endothelial cell apoptotic process |
GO_0060216 | Biological process | definitive hemopoiesis |
GO_0051591 | Biological process | response to cAMP |
GO_0050728 | Biological process | negative regulation of inflammatory response |
GO_0043066 | Biological process | negative regulation of apoptotic process |
GO_0001938 | Biological process | positive regulation of endothelial cell proliferation |
GO_0034446 | Biological process | substrate adhesion-dependent cell spreading |
GO_0048014 | Biological process | Tie signaling pathway |
GO_0001666 | Biological process | response to hypoxia |
GO_0001935 | Biological process | endothelial cell proliferation |
GO_0051894 | Biological process | positive regulation of focal adhesion assembly |
GO_0051897 | Biological process | positive regulation of phosphatidylinositol 3-kinase/protein kinase B signal transduction |
GO_0072012 | Biological process | glomerulus vasculature development |
GO_0035025 | Biological process | positive regulation of Rho protein signal transduction |
GO_0070374 | Biological process | positive regulation of ERK1 and ERK2 cascade |
GO_0032878 | Biological process | regulation of establishment or maintenance of cell polarity |
GO_0005902 | Cellular component | microvillus |
GO_0016324 | Cellular component | apical plasma membrane |
GO_0043235 | Cellular component | receptor complex |
GO_0005576 | Cellular component | extracellular region |
GO_0005886 | Cellular component | plasma membrane |
GO_0034451 | Cellular component | centriolar satellite |
GO_0045121 | Cellular component | membrane raft |
GO_0009925 | Cellular component | basal plasma membrane |
GO_0005884 | Cellular component | actin filament |
GO_0005925 | Cellular component | focal adhesion |
GO_0005911 | Cellular component | cell-cell junction |
GO_0009986 | Cellular component | cell surface |
GO_0001725 | Cellular component | stress fiber |
GO_0005737 | Cellular component | cytoplasm |
GO_0016323 | Cellular component | basolateral plasma membrane |
GO_0038023 | Molecular function | signaling receptor activity |
GO_0042802 | Molecular function | identical protein binding |
GO_0004714 | Molecular function | transmembrane receptor protein tyrosine kinase activity |
GO_0004672 | Molecular function | protein kinase activity |
GO_0019838 | Molecular function | growth factor binding |
GO_0005524 | Molecular function | ATP binding |
GO_0005515 | Molecular function | protein binding |
Gene name | TEK |
Protein name | TEK receptor tyrosine kinase Angiopoietin-1 receptor (EC 2.7.10.1) (Endothelial tyrosine kinase) (Tunica interna endothelial cell kinase) (Tyrosine kinase with Ig and EGF homology domains-2) (Tyrosine-protein kinase receptor TEK) (Tyrosine-protein kinase receptor TIE-2) (hTIE2) (p140 TEK) (CD antigen CD202b) Mutant TEK tyrosine kinase Soluble TIE2 variant 1 Soluble TIE2 variant 2 (TEK receptor tyrosine kinase) |
Synonyms | TIE2 VMCM1 VMCM |
Description | FUNCTION: Tyrosine-protein kinase that acts as a cell-surface receptor for ANGPT1, ANGPT2 and ANGPT4 and regulates angiogenesis, endothelial cell survival, proliferation, migration, adhesion and cell spreading, reorganization of the actin cytoskeleton, but also maintenance of vascular quiescence. Has anti-inflammatory effects by preventing the leakage of pro-inflammatory plasma proteins and leukocytes from blood vessels. Required for normal angiogenesis and heart development during embryogenesis. Required for post-natal hematopoiesis. After birth, activates or inhibits angiogenesis, depending on the context. Inhibits angiogenesis and promotes vascular stability in quiescent vessels, where endothelial cells have tight contacts. In quiescent vessels, ANGPT1 oligomers recruit TEK to cell-cell contacts, forming complexes with TEK molecules from adjoining cells, and this leads to preferential activation of phosphatidylinositol 3-kinase and the AKT1 signaling cascades. In migrating endothelial cells that lack cell-cell adhesions, ANGT1 recruits TEK to contacts with the extracellular matrix, leading to the formation of focal adhesion complexes, activation of PTK2/FAK and of the downstream kinases MAPK1/ERK2 and MAPK3/ERK1, and ultimately to the stimulation of sprouting angiogenesis. ANGPT1 signaling triggers receptor dimerization and autophosphorylation at specific tyrosine residues that then serve as binding sites for scaffold proteins and effectors. Signaling is modulated by ANGPT2 that has lower affinity for TEK, can promote TEK autophosphorylation in the absence of ANGPT1, but inhibits ANGPT1-mediated signaling by competing for the same binding site. Signaling is also modulated by formation of heterodimers with TIE1, and by proteolytic processing that gives rise to a soluble TEK extracellular domain. The soluble extracellular domain modulates signaling by functioning as decoy receptor for angiopoietins. TEK phosphorylates DOK2, GRB7, GRB14, PIK3R1; SHC1 and TIE1. . |
Accessions | D9MND2 D9MND0 ENST00000615002.4 ENST00000406359.8 [Q02763-2] ENST00000380036.10 [Q02763-1] B5A953 E5RIV9 B5A954 ENST00000519097.5 [Q02763-3] Q02763 ENST00000519080.1 |