Name | Number of supported studies | Average coverage | |
---|---|---|---|
endothelial cell | 4 studies | 22% ± 3% | |
epithelial cell | 4 studies | 26% ± 13% | |
macrophage | 4 studies | 18% ± 2% | |
GABAergic neuron | 3 studies | 30% ± 4% | |
glutamatergic neuron | 3 studies | 34% ± 4% | |
microglial cell | 3 studies | 21% ± 4% | |
dendritic cell | 3 studies | 23% ± 8% | |
astrocyte | 3 studies | 21% ± 4% | |
oligodendrocyte | 3 studies | 21% ± 3% |
Name | Number of supported studies | Average coverage | |
---|---|---|---|
brain | 4 studies | 28% ± 2% |
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
esophagus | 100% | 1620.32 | 1445 / 1445 | 100% | 20.24 | 183 / 183 |
ovary | 100% | 2436.72 | 180 / 180 | 100% | 15.33 | 429 / 430 |
brain | 100% | 1218.43 | 2638 / 2642 | 100% | 22.60 | 703 / 705 |
lung | 100% | 1550.71 | 576 / 578 | 100% | 18.58 | 1152 / 1155 |
prostate | 100% | 1576.96 | 245 / 245 | 99% | 24.45 | 498 / 502 |
breast | 100% | 2062.70 | 459 / 459 | 99% | 28.62 | 1109 / 1118 |
thymus | 100% | 1601.86 | 653 / 653 | 98% | 21.91 | 595 / 605 |
stomach | 100% | 1358.36 | 359 / 359 | 98% | 18.10 | 280 / 286 |
intestine | 100% | 1959.46 | 966 / 966 | 98% | 16.79 | 515 / 527 |
pancreas | 99% | 955.21 | 324 / 328 | 98% | 16.13 | 175 / 178 |
bladder | 100% | 2055.67 | 21 / 21 | 97% | 14.76 | 488 / 504 |
uterus | 100% | 2359.39 | 170 / 170 | 96% | 17.54 | 442 / 459 |
kidney | 100% | 955.29 | 89 / 89 | 96% | 18.93 | 866 / 901 |
adrenal gland | 100% | 3092.67 | 258 / 258 | 96% | 14.83 | 221 / 230 |
skin | 100% | 1684.07 | 1809 / 1809 | 91% | 14.60 | 429 / 472 |
liver | 100% | 866.09 | 225 / 226 | 86% | 9.67 | 350 / 406 |
blood vessel | 100% | 2164.52 | 1335 / 1335 | 0% | 0 | 0 / 0 |
muscle | 100% | 1559.64 | 803 / 803 | 0% | 0 | 0 / 0 |
spleen | 100% | 1445.68 | 241 / 241 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 100% | 4.86 | 1 / 1 |
adipose | 100% | 2223.84 | 1203 / 1204 | 0% | 0 | 0 / 0 |
lymph node | 0% | 0 | 0 / 0 | 97% | 15.74 | 28 / 29 |
heart | 96% | 843.40 | 826 / 861 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 96% | 13.92 | 43 / 45 |
eye | 0% | 0 | 0 / 0 | 85% | 11.20 | 68 / 80 |
peripheral blood | 69% | 657.43 | 640 / 929 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
GO_0097009 | Biological process | energy homeostasis |
GO_0042542 | Biological process | response to hydrogen peroxide |
GO_0045786 | Biological process | negative regulation of cell cycle |
GO_0006642 | Biological process | triglyceride mobilization |
GO_0051097 | Biological process | negative regulation of helicase activity |
GO_0032071 | Biological process | regulation of endodeoxyribonuclease activity |
GO_0000183 | Biological process | rDNA heterochromatin formation |
GO_0030225 | Biological process | macrophage differentiation |
GO_2000773 | Biological process | negative regulation of cellular senescence |
GO_0006346 | Biological process | DNA methylation-dependent heterochromatin formation |
GO_0046015 | Biological process | regulation of transcription by glucose |
GO_0009267 | Biological process | cellular response to starvation |
GO_0051152 | Biological process | positive regulation of smooth muscle cell differentiation |
GO_2000655 | Biological process | negative regulation of cellular response to testosterone stimulus |
GO_0032088 | Biological process | negative regulation of NF-kappaB transcription factor activity |
GO_2000757 | Biological process | negative regulation of peptidyl-lysine acetylation |
GO_0045722 | Biological process | positive regulation of gluconeogenesis |
GO_0043066 | Biological process | negative regulation of apoptotic process |
GO_0007346 | Biological process | regulation of mitotic cell cycle |
GO_0060766 | Biological process | negative regulation of androgen receptor signaling pathway |
GO_1902166 | Biological process | negative regulation of intrinsic apoptotic signaling pathway in response to DNA damage by p53 class mediator |
GO_0051897 | Biological process | positive regulation of phosphatidylinositol 3-kinase/protein kinase B signal transduction |
GO_0006979 | Biological process | response to oxidative stress |
GO_0031393 | Biological process | negative regulation of prostaglandin biosynthetic process |
GO_0043161 | Biological process | proteasome-mediated ubiquitin-dependent protein catabolic process |
GO_0070301 | Biological process | cellular response to hydrogen peroxide |
GO_2000480 | Biological process | negative regulation of cAMP-dependent protein kinase activity |
GO_0050872 | Biological process | white fat cell differentiation |
GO_1901797 | Biological process | negative regulation of signal transduction by p53 class mediator |
GO_0001525 | Biological process | angiogenesis |
GO_0010868 | Biological process | negative regulation of triglyceride biosynthetic process |
GO_0045348 | Biological process | positive regulation of MHC class II biosynthetic process |
GO_0090335 | Biological process | regulation of brown fat cell differentiation |
GO_0010629 | Biological process | negative regulation of gene expression |
GO_0045766 | Biological process | positive regulation of angiogenesis |
GO_0070914 | Biological process | UV-damage excision repair |
GO_0032007 | Biological process | negative regulation of TOR signaling |
GO_0045599 | Biological process | negative regulation of fat cell differentiation |
GO_0034391 | Biological process | regulation of smooth muscle cell apoptotic process |
GO_0071900 | Biological process | regulation of protein serine/threonine kinase activity |
GO_0031507 | Biological process | heterochromatin formation |
GO_0035358 | Biological process | regulation of peroxisome proliferator activated receptor signaling pathway |
GO_1900034 | Biological process | regulation of cellular response to heat |
GO_0051658 | Biological process | maintenance of nucleus location |
GO_0042981 | Biological process | regulation of apoptotic process |
GO_0016239 | Biological process | positive regulation of macroautophagy |
GO_0010883 | Biological process | regulation of lipid storage |
GO_0000731 | Biological process | DNA synthesis involved in DNA repair |
GO_0042595 | Biological process | behavioral response to starvation |
GO_0071479 | Biological process | cellular response to ionizing radiation |
GO_2000481 | Biological process | positive regulation of cAMP-dependent protein kinase activity |
GO_0043124 | Biological process | negative regulation of canonical NF-kappaB signal transduction |
GO_0043518 | Biological process | negative regulation of DNA damage response, signal transduction by p53 class mediator |
GO_1904179 | Biological process | positive regulation of adipose tissue development |
GO_0042149 | Biological process | cellular response to glucose starvation |
GO_0016567 | Biological process | protein ubiquitination |
GO_0055089 | Biological process | fatty acid homeostasis |
GO_0001542 | Biological process | ovulation from ovarian follicle |
GO_0043536 | Biological process | positive regulation of blood vessel endothelial cell migration |
GO_0140861 | Biological process | DNA repair-dependent chromatin remodeling |
GO_0045739 | Biological process | positive regulation of DNA repair |
GO_1990830 | Biological process | cellular response to leukemia inhibitory factor |
GO_0042771 | Biological process | intrinsic apoptotic signaling pathway in response to DNA damage by p53 class mediator |
GO_0006476 | Biological process | protein deacetylation |
GO_0001934 | Biological process | positive regulation of protein phosphorylation |
GO_0031648 | Biological process | protein destabilization |
GO_0043433 | Biological process | negative regulation of DNA-binding transcription factor activity |
GO_0001678 | Biological process | intracellular glucose homeostasis |
GO_1902237 | Biological process | positive regulation of endoplasmic reticulum stress-induced intrinsic apoptotic signaling pathway |
GO_0010824 | Biological process | regulation of centrosome duplication |
GO_0033210 | Biological process | leptin-mediated signaling pathway |
GO_0007517 | Biological process | muscle organ development |
GO_0002821 | Biological process | positive regulation of adaptive immune response |
GO_0046628 | Biological process | positive regulation of insulin receptor signaling pathway |
GO_0060907 | Biological process | positive regulation of macrophage cytokine production |
GO_0008284 | Biological process | positive regulation of cell population proliferation |
GO_0043524 | Biological process | negative regulation of neuron apoptotic process |
GO_0043065 | Biological process | positive regulation of apoptotic process |
GO_2000781 | Biological process | positive regulation of double-strand break repair |
GO_0045892 | Biological process | negative regulation of DNA-templated transcription |
GO_0042127 | Biological process | regulation of cell population proliferation |
GO_0071456 | Biological process | cellular response to hypoxia |
GO_0045944 | Biological process | positive regulation of transcription by RNA polymerase II |
GO_0006325 | Biological process | chromatin organization |
GO_2000111 | Biological process | positive regulation of macrophage apoptotic process |
GO_0007283 | Biological process | spermatogenesis |
GO_0000012 | Biological process | single strand break repair |
GO_2000774 | Biological process | positive regulation of cellular senescence |
GO_0030512 | Biological process | negative regulation of transforming growth factor beta receptor signaling pathway |
GO_0044321 | Biological process | response to leptin |
GO_0006974 | Biological process | DNA damage response |
GO_0106230 | Biological process | protein depropionylation |
GO_0032868 | Biological process | response to insulin |
GO_1902176 | Biological process | negative regulation of oxidative stress-induced intrinsic apoptotic signaling pathway |
GO_0010906 | Biological process | regulation of glucose metabolic process |
GO_0000720 | Biological process | pyrimidine dimer repair by nucleotide-excision repair |
GO_0090400 | Biological process | stress-induced premature senescence |
GO_0000122 | Biological process | negative regulation of transcription by RNA polymerase II |
GO_0043280 | Biological process | positive regulation of cysteine-type endopeptidase activity involved in apoptotic process |
GO_0018394 | Biological process | peptidyl-lysine acetylation |
GO_0042326 | Biological process | negative regulation of phosphorylation |
GO_0032922 | Biological process | circadian regulation of gene expression |
GO_0051898 | Biological process | negative regulation of phosphatidylinositol 3-kinase/protein kinase B signal transduction |
GO_0071356 | Biological process | cellular response to tumor necrosis factor |
GO_0001938 | Biological process | positive regulation of endothelial cell proliferation |
GO_0010875 | Biological process | positive regulation of cholesterol efflux |
GO_0070857 | Biological process | regulation of bile acid biosynthetic process |
GO_0007179 | Biological process | transforming growth factor beta receptor signaling pathway |
GO_1901984 | Biological process | negative regulation of protein acetylation |
GO_0035356 | Biological process | intracellular triglyceride homeostasis |
GO_0042632 | Biological process | cholesterol homeostasis |
GO_0035098 | Cellular component | ESC/E(Z) complex |
GO_0016605 | Cellular component | PML body |
GO_0005634 | Cellular component | nucleus |
GO_0005730 | Cellular component | nucleolus |
GO_0005654 | Cellular component | nucleoplasm |
GO_0001650 | Cellular component | fibrillar center |
GO_0061773 | Cellular component | eNoSc complex |
GO_0005739 | Cellular component | mitochondrion |
GO_0000792 | Cellular component | heterochromatin |
GO_0000791 | Cellular component | euchromatin |
GO_0005737 | Cellular component | cytoplasm |
GO_0033553 | Cellular component | rDNA heterochromatin |
GO_0000785 | Cellular component | chromatin |
GO_0005677 | Cellular component | chromatin silencing complex |
GO_0005637 | Cellular component | nuclear inner membrane |
GO_0005635 | Cellular component | nuclear envelope |
GO_0005829 | Cellular component | cytosol |
GO_0140297 | Molecular function | DNA-binding transcription factor binding |
GO_0042802 | Molecular function | identical protein binding |
GO_0106231 | Molecular function | protein-propionyllysine depropionylase activity |
GO_0046970 | Molecular function | NAD-dependent histone H4K16 deacetylase activity |
GO_0140937 | Molecular function | histone H4K12 deacetylase activity |
GO_1990404 | Molecular function | NAD+-protein ADP-ribosyltransferase activity |
GO_0000978 | Molecular function | RNA polymerase II cis-regulatory region sequence-specific DNA binding |
GO_1990841 | Molecular function | promoter-specific chromatin binding |
GO_0042393 | Molecular function | histone binding |
GO_0070577 | Molecular function | lysine-acetylated histone binding |
GO_0160012 | Molecular function | NAD-dependent histone decrotonylase activity |
GO_0046969 | Molecular function | NAD-dependent histone H3K9 deacetylase activity |
GO_0046872 | Molecular function | metal ion binding |
GO_0032041 | Molecular function | NAD-dependent histone H3K14 deacetylase activity |
GO_1990254 | Molecular function | keratin filament binding |
GO_0019899 | Molecular function | enzyme binding |
GO_0019213 | Molecular function | deacetylase activity |
GO_0003950 | Molecular function | NAD+ ADP-ribosyltransferase activity |
GO_0004407 | Molecular function | histone deacetylase activity |
GO_0017136 | Molecular function | NAD-dependent histone deacetylase activity |
GO_0070403 | Molecular function | NAD+ binding |
GO_0034979 | Molecular function | NAD-dependent protein lysine deacetylase activity |
GO_0002039 | Molecular function | p53 binding |
GO_0005515 | Molecular function | protein binding |
GO_0033558 | Molecular function | protein lysine deacetylase activity |
GO_0016922 | Molecular function | nuclear receptor binding |
GO_0043425 | Molecular function | bHLH transcription factor binding |
GO_0141050 | Molecular function | histone H3K deacetylase activity |
GO_0003714 | Molecular function | transcription corepressor activity |
GO_0051019 | Molecular function | mitogen-activated protein kinase binding |
GO_0003713 | Molecular function | transcription coactivator activity |
GO_0043398 | Molecular function | HLH domain binding |
Gene name | SIRT1 |
Protein name | Sirtuin 1 (cDNA FLJ90324 fis, clone NT2RP2001817, highly similar to NAD-dependent deacetylase sirtuin-1) Sirtuin 1 NAD-dependent protein deacetylase sirtuin-1 (hSIRT1) (EC 2.3.1.286) (NAD-dependent protein deacylase sirtuin-1) (EC 2.3.1.-) (Regulatory protein SIR2 homolog 1) (SIR2-like protein 1) (hSIR2) [Cleaved into: SirtT1 75 kDa fragment (75SirT1)] |
Synonyms | SIR2L1 |
Description | FUNCTION: NAD-dependent protein deacetylase that links transcriptional regulation directly to intracellular energetics and participates in the coordination of several separated cellular functions such as cell cycle, response to DNA damage, metabolism, apoptosis and autophagy . Can modulate chromatin function through deacetylation of histones and can promote alterations in the methylation of histones and DNA, leading to transcriptional repression . Deacetylates a broad range of transcription factors and coregulators, thereby regulating target gene expression positively and negatively . Serves as a sensor of the cytosolic ratio of NAD(+)/NADH which is altered by glucose deprivation and metabolic changes associated with caloric restriction . Is essential in skeletal muscle cell differentiation and in response to low nutrients mediates the inhibitory effect on skeletal myoblast differentiation which also involves 5'-AMP-activated protein kinase (AMPK) and nicotinamide phosphoribosyltransferase (NAMPT) (By similarity). Component of the eNoSC (energy-dependent nucleolar silencing) complex, a complex that mediates silencing of rDNA in response to intracellular energy status and acts by recruiting histone-modifying enzymes . The eNoSC complex is able to sense the energy status of cell: upon glucose starvation, elevation of NAD(+)/NADP(+) ratio activates SIRT1, leading to histone H3 deacetylation followed by dimethylation of H3 at 'Lys-9' (H3K9me2) by SUV39H1 and the formation of silent chromatin in the rDNA locus . Deacetylates 'Lys-266' of SUV39H1, leading to its activation . Inhibits skeletal muscle differentiation by deacetylating PCAF and MYOD1 . Deacetylates H2A and 'Lys-26' of H1-4 . Deacetylates 'Lys-16' of histone H4 (in vitro). Involved in NR0B2/SHP corepression function through chromatin remodeling: Recruited to LRH1 target gene promoters by NR0B2/SHP thereby stimulating histone H3 and H4 deacetylation leading to transcriptional repression . Proposed to contribute to genomic integrity via positive regulation of telomere length; however, reports on localization to pericentromeric heterochromatin are conflicting (By similarity). Proposed to play a role in constitutive heterochromatin (CH) formation and/or maintenance through regulation of the available pool of nuclear SUV39H1 . Upon oxidative/metabolic stress decreases SUV39H1 degradation by inhibiting SUV39H1 polyubiquitination by MDM2 . This increase in SUV39H1 levels enhances SUV39H1 turnover in CH, which in turn seems to accelerate renewal of the heterochromatin which correlates with greater genomic integrity during stress response . Deacetylates 'Lys-382' of p53/TP53 and impairs its ability to induce transcription-dependent proapoptotic program and modulate cell senescence . Deacetylates TAF1B and thereby represses rDNA transcription by the RNA polymerase I (By similarity). Deacetylates MYC, promotes the association of MYC with MAX and decreases MYC stability leading to compromised transformational capability . Deacetylates FOXO3 in response to oxidative stress thereby increasing its ability to induce cell cycle arrest and resistance to oxidative stress but inhibiting FOXO3-mediated induction of apoptosis transcriptional activity; also leading to FOXO3 ubiquitination and protesomal degradation . Appears to have a similar effect on MLLT7/FOXO4 in regulation of transcriptional activity and apoptosis . Deacetylates DNMT1; thereby impairs DNMT1 methyltransferase-independent transcription repressor activity, modulates DNMT1 cell cycle regulatory function and DNMT1-mediated gene silencing . Deacetylates RELA/NF-kappa-B p65 thereby inhibiting its transactivating potential and augments apoptosis in response to TNF-alpha . Deacetylates HIF1A, KAT5/TIP60, RB1 and HIC1 . Deacetylates FOXO1 resulting in its nuclear retention and enhancement of its transcriptional activity leading to increased gluconeogenesis in liver . Inhibits E2F1 transcriptional activity and apoptotic function, possibly by deacetylation . Involved in HES1- and HEY2-mediated transcriptional repression . In cooperation with MYCN seems to be involved in transcriptional repression of DUSP6/MAPK3 leading to MYCN stabilization by phosphorylation at 'Ser-62' . Deacetylates MEF2D . Required for antagonist-mediated transcription suppression of AR-dependent genes which may be linked to local deacetylation of histone H3 . Represses HNF1A-mediated transcription (By similarity). Required for the repression of ESRRG by CREBZF . Deacetylates NR1H3 and NR1H2 and deacetylation of NR1H3 at 'Lys-434' positively regulates transcription of NR1H3:RXR target genes, promotes NR1H3 proteasomal degradation and results in cholesterol efflux; a promoter clearing mechanism after reach round of transcription is proposed . Involved in lipid metabolism: deacetylates LPIN1, thereby inhibiting diacylglycerol synthesis . Implicated in regulation of adipogenesis and fat mobilization in white adipocytes by repression of PPARG which probably involves association with NCOR1 and SMRT/NCOR2 (By similarity). Deacetylates p300/EP300 and PRMT1 (By similarity). Deacetylates ACSS2 leading to its activation, and HMGCS1 deacetylation . Involved in liver and muscle metabolism. Through deacetylation and activation of PPARGC1A is required to activate fatty acid oxidation in skeletal muscle under low-glucose conditions and is involved in glucose homeostasis . Involved in regulation of PPARA and fatty acid beta-oxidation in liver. Involved in positive regulation of insulin secretion in pancreatic beta cells in response to glucose; the function seems to imply transcriptional repression of UCP2. Proposed to deacetylate IRS2 thereby facilitating its insulin-induced tyrosine phosphorylation. Deacetylates SREBF1 isoform SREBP-1C thereby decreasing its stability and transactivation in lipogenic gene expression . Involved in DNA damage response by repressing genes which are involved in DNA repair, such as XPC and TP73, deacetylating XRCC6/Ku70, and facilitating recruitment of additional factors to sites of damaged DNA, such as SIRT1-deacetylated NBN can recruit ATM to initiate DNA repair and SIRT1-deacetylated XPA interacts with RPA2 . Also involved in DNA repair of DNA double-strand breaks by homologous recombination and specifically single-strand annealing independently of XRCC6/Ku70 and NBN . Promotes DNA double-strand breaks by mediating deacetylation of SIRT6 . Transcriptional suppression of XPC probably involves an E2F4:RBL2 suppressor complex and protein kinase B (AKT) signaling. Transcriptional suppression of TP73 probably involves E2F4 and PCAF. Deacetylates WRN thereby regulating its helicase and exonuclease activities and regulates WRN nuclear translocation in response to DNA damage . Deacetylates APEX1 at 'Lys-6' and 'Lys-7' and stimulates cellular AP endonuclease activity by promoting the association of APEX1 to XRCC1 . Catalyzes deacetylation of ERCC4/XPF, thereby impairing interaction with ERCC1 and nucleotide excision repair (NER) . Increases p53/TP53-mediated transcription-independent apoptosis by blocking nuclear translocation of cytoplasmic p53/TP53 and probably redirecting it to mitochondria. Deacetylates XRCC6/Ku70 at 'Lys-539' and 'Lys-542' causing it to sequester BAX away from mitochondria thereby inhibiting stress-induced apoptosis. Is involved in autophagy, presumably by deacetylating ATG5, ATG7 and MAP1LC3B/ATG8 . Deacetylates AKT1 which leads to enhanced binding of AKT1 and PDK1 to PIP3 and promotes their activation . Proposed to play role in regulation of STK11/LBK1-dependent AMPK signaling pathways implicated in cellular senescence which seems to involve the regulation of the acetylation status of STK11/LBK1. Can deacetylate STK11/LBK1 and thereby increase its activity, cytoplasmic localization and association with STRAD; however, the relevance of such activity in normal cells is unclear . In endothelial cells is shown to inhibit STK11/LBK1 activity and to promote its degradation. Deacetylates SMAD7 at 'Lys-64' and 'Lys-70' thereby promoting its degradation. Deacetylates CIITA and augments its MHC class II transactivation and contributes to its stability . Deacetylates MECOM/EVI1 . Deacetylates PML at 'Lys-487' and this deacetylation promotes PML control of PER2 nuclear localization . During the neurogenic transition, represses selective NOTCH1-target genes through histone deacetylation in a BCL6-dependent manner and leading to neuronal differentiation. Regulates the circadian expression of several core clock genes, including BMAL1, RORC, PER2 and CRY1 and plays a critical role in maintaining a controlled rhythmicity in histone acetylation, thereby contributing to circadian chromatin remodeling . Deacetylates BMAL1 and histones at the circadian gene promoters in order to facilitate repression by inhibitory components of the circadian oscillator (By similarity). Deacetylates PER2, facilitating its ubiquitination and degradation by the proteasome (By similarity). Protects cardiomyocytes against palmitate-induced apoptosis (By similarity). Deacetylates XBP1 isoform 2; deacetylation decreases protein stability of XBP1 isoform 2 and inhibits its transcriptional activity . Deacetylates PCK1 and directs its activity toward phosphoenolpyruvate production promoting gluconeogenesis . Involved in the CCAR2-mediated regulation of PCK1 and NR1D1 . Deacetylates CTNB1 at 'Lys-49' . In POMC (pro-opiomelanocortin) neurons, required for leptin-induced activation of PI3K signaling (By similarity). In addition to protein deacetylase activity, also acts as a protein-lysine deacylase by mediating protein depropionylation and decrotonylation . Mediates depropionylation of Osterix (SP7) (By similarity). Catalyzes decrotonylation of histones; it however does not represent a major histone decrotonylase . Deacetylates SOX9; promoting SOX9 nuclear localization and transactivation activity (By similarity). Involved in the regulation of centrosome duplication. Deacetylates CENATAC in G1 phase, allowing for SASS6 accumulation on the centrosome and subsequent procentriole assembly . Deacetylates NDC80/HEC1 . .; FUNCTION: [Isoform 2]: Deacetylates 'Lys-382' of p53/TP53, however with lower activity than isoform 1. In combination, the two isoforms exert an additive effect. Isoform 2 regulates p53/TP53 expression and cellular stress response and is in turn repressed by p53/TP53 presenting a SIRT1 isoform-dependent auto-regulatory loop. .; FUNCTION: [SirtT1 75 kDa fragment]: Catalytically inactive 75SirT1 may be involved in regulation of apoptosis. May be involved in protecting chondrocytes from apoptotic death by associating with cytochrome C and interfering with apoptosome assembly. .; FUNCTION: (Microbial infection) In case of HIV-1 infection, interacts with and deacetylates the viral Tat protein. The viral Tat protein inhibits SIRT1 deacetylation activity toward RELA/NF-kappa-B p65, thereby potentiates its transcriptional activity and SIRT1 is proposed to contribute to T-cell hyperactivation during infection. . |
Accessions | E9PC49 ENST00000406900.5 ENST00000403579.1 Q96EB6 ENST00000212015.11 [Q96EB6-1] I1Y8W7 B0QZ35 ENST00000432464.5 |