Name | Number of supported studies | Average coverage | |
---|---|---|---|
epithelial cell | 5 studies | 31% ± 13% | |
astrocyte | 4 studies | 23% ± 6% | |
hematopoietic precursor cell | 3 studies | 22% ± 3% | |
endothelial cell | 3 studies | 23% ± 9% | |
dendritic cell | 3 studies | 25% ± 6% | |
intestinal crypt stem cell | 3 studies | 22% ± 4% | |
goblet cell | 3 studies | 23% ± 4% |
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
thymus | 100% | 1778.09 | 652 / 653 | 100% | 38.03 | 602 / 605 |
breast | 100% | 2537.42 | 459 / 459 | 97% | 52.84 | 1087 / 1118 |
prostate | 100% | 2009.89 | 245 / 245 | 97% | 39.68 | 486 / 502 |
uterus | 100% | 2572.82 | 170 / 170 | 96% | 55.18 | 441 / 459 |
skin | 100% | 3581.19 | 1809 / 1809 | 96% | 49.10 | 451 / 472 |
intestine | 100% | 1952.40 | 966 / 966 | 95% | 61.65 | 503 / 527 |
bladder | 100% | 2358.90 | 21 / 21 | 94% | 50.76 | 473 / 504 |
ovary | 100% | 2755.63 | 180 / 180 | 93% | 69.12 | 400 / 430 |
esophagus | 100% | 1961.75 | 1443 / 1445 | 93% | 47.38 | 170 / 183 |
lung | 99% | 1615.52 | 573 / 578 | 93% | 42.42 | 1078 / 1155 |
stomach | 99% | 1419.45 | 357 / 359 | 93% | 54.48 | 266 / 286 |
kidney | 99% | 1199.56 | 88 / 89 | 93% | 28.93 | 839 / 901 |
pancreas | 98% | 1114.71 | 323 / 328 | 93% | 28.41 | 166 / 178 |
liver | 100% | 1007.75 | 225 / 226 | 68% | 17.13 | 277 / 406 |
adrenal gland | 99% | 1567.92 | 256 / 258 | 64% | 11.78 | 147 / 230 |
brain | 65% | 417.25 | 1719 / 2642 | 95% | 42.45 | 673 / 705 |
adipose | 100% | 2158.45 | 1204 / 1204 | 0% | 0 | 0 / 0 |
lymph node | 0% | 0 | 0 / 0 | 100% | 70.13 | 29 / 29 |
spleen | 100% | 2766.55 | 241 / 241 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 100% | 11.57 | 1 / 1 |
blood vessel | 100% | 1778.92 | 1334 / 1335 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 99% | 45.97 | 79 / 80 |
tonsil | 0% | 0 | 0 / 0 | 98% | 74.07 | 44 / 45 |
heart | 96% | 791.72 | 824 / 861 | 0% | 0 | 0 / 0 |
peripheral blood | 94% | 2482.08 | 875 / 929 | 0% | 0 | 0 / 0 |
muscle | 85% | 807.76 | 686 / 803 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
GO_0002326 | Biological process | B cell lineage commitment |
GO_0043504 | Biological process | mitochondrial DNA repair |
GO_0048147 | Biological process | negative regulation of fibroblast proliferation |
GO_0051097 | Biological process | negative regulation of helicase activity |
GO_0009651 | Biological process | response to salt stress |
GO_0043516 | Biological process | regulation of DNA damage response, signal transduction by p53 class mediator |
GO_0010332 | Biological process | response to gamma radiation |
GO_0006289 | Biological process | nucleotide-excision repair |
GO_0006978 | Biological process | DNA damage response, signal transduction by p53 class mediator resulting in transcription of p21 class mediator |
GO_0035264 | Biological process | multicellular organism growth |
GO_2000647 | Biological process | negative regulation of stem cell proliferation |
GO_0051262 | Biological process | protein tetramerization |
GO_0060218 | Biological process | hematopoietic stem cell differentiation |
GO_0051402 | Biological process | neuron apoptotic process |
GO_0032211 | Biological process | negative regulation of telomere maintenance via telomerase |
GO_0090403 | Biological process | oxidative stress-induced premature senescence |
GO_0043066 | Biological process | negative regulation of apoptotic process |
GO_0045861 | Biological process | negative regulation of proteolysis |
GO_2000379 | Biological process | positive regulation of reactive oxygen species metabolic process |
GO_0050821 | Biological process | protein stabilization |
GO_1902895 | Biological process | positive regulation of miRNA transcription |
GO_0021549 | Biological process | cerebellum development |
GO_0016032 | Biological process | viral process |
GO_0043153 | Biological process | entrainment of circadian clock by photoperiod |
GO_0000423 | Biological process | mitophagy |
GO_0006914 | Biological process | autophagy |
GO_0070266 | Biological process | necroptotic process |
GO_0097252 | Biological process | oligodendrocyte apoptotic process |
GO_1900119 | Biological process | positive regulation of execution phase of apoptosis |
GO_0051726 | Biological process | regulation of cell cycle |
GO_0072089 | Biological process | stem cell proliferation |
GO_0090200 | Biological process | positive regulation of release of cytochrome c from mitochondria |
GO_0002244 | Biological process | hematopoietic progenitor cell differentiation |
GO_0008285 | Biological process | negative regulation of cell population proliferation |
GO_0071466 | Biological process | cellular response to xenobiotic stimulus |
GO_0002931 | Biological process | response to ischemia |
GO_0001701 | Biological process | in utero embryonic development |
GO_0034644 | Biological process | cellular response to UV |
GO_0006357 | Biological process | regulation of transcription by RNA polymerase II |
GO_0006302 | Biological process | double-strand break repair |
GO_0070245 | Biological process | positive regulation of thymocyte apoptotic process |
GO_0010628 | Biological process | positive regulation of gene expression |
GO_2001244 | Biological process | positive regulation of intrinsic apoptotic signaling pathway |
GO_0060333 | Biological process | type II interferon-mediated signaling pathway |
GO_0007369 | Biological process | gastrulation |
GO_0070242 | Biological process | thymocyte apoptotic process |
GO_0071480 | Biological process | cellular response to gamma radiation |
GO_0072593 | Biological process | reactive oxygen species metabolic process |
GO_0033077 | Biological process | T cell differentiation in thymus |
GO_0097193 | Biological process | intrinsic apoptotic signaling pathway |
GO_1901525 | Biological process | negative regulation of mitophagy |
GO_0042981 | Biological process | regulation of apoptotic process |
GO_1902108 | Biological process | regulation of mitochondrial membrane permeability involved in apoptotic process |
GO_0048144 | Biological process | fibroblast proliferation |
GO_0006606 | Biological process | protein import into nucleus |
GO_0034103 | Biological process | regulation of tissue remodeling |
GO_0006355 | Biological process | regulation of DNA-templated transcription |
GO_0006977 | Biological process | DNA damage response, signal transduction by p53 class mediator resulting in cell cycle arrest |
GO_0002309 | Biological process | T cell proliferation involved in immune response |
GO_0031571 | Biological process | mitotic G1 DNA damage checkpoint signaling |
GO_0009299 | Biological process | mRNA transcription |
GO_0071479 | Biological process | cellular response to ionizing radiation |
GO_0009303 | Biological process | rRNA transcription |
GO_0007405 | Biological process | neuroblast proliferation |
GO_0007406 | Biological process | negative regulation of neuroblast proliferation |
GO_0030308 | Biological process | negative regulation of cell growth |
GO_1902749 | Biological process | regulation of cell cycle G2/M phase transition |
GO_1902253 | Biological process | regulation of intrinsic apoptotic signaling pathway by p53 class mediator |
GO_0042149 | Biological process | cellular response to glucose starvation |
GO_0070059 | Biological process | intrinsic apoptotic signaling pathway in response to endoplasmic reticulum stress |
GO_0007265 | Biological process | Ras protein signal transduction |
GO_1903451 | Biological process | negative regulation of G1 to G0 transition |
GO_0048568 | Biological process | embryonic organ development |
GO_0060411 | Biological process | cardiac septum morphogenesis |
GO_0072717 | Biological process | cellular response to actinomycin D |
GO_0045899 | Biological process | positive regulation of RNA polymerase II transcription preinitiation complex assembly |
GO_0042771 | Biological process | intrinsic apoptotic signaling pathway in response to DNA damage by p53 class mediator |
GO_0090399 | Biological process | replicative senescence |
GO_1903799 | Biological process | negative regulation of miRNA processing |
GO_1904024 | Biological process | negative regulation of glucose catabolic process to lactate via pyruvate |
GO_0008156 | Biological process | negative regulation of DNA replication |
GO_0045893 | Biological process | positive regulation of DNA-templated transcription |
GO_0060253 | Biological process | negative regulation of glial cell proliferation |
GO_0072332 | Biological process | intrinsic apoptotic signaling pathway by p53 class mediator |
GO_0046677 | Biological process | response to antibiotic |
GO_0001756 | Biological process | somitogenesis |
GO_0001836 | Biological process | release of cytochrome c from mitochondria |
GO_0035794 | Biological process | positive regulation of mitochondrial membrane permeability |
GO_1905856 | Biological process | negative regulation of pentose-phosphate shunt |
GO_0002360 | Biological process | T cell lineage commitment |
GO_0043065 | Biological process | positive regulation of apoptotic process |
GO_0045892 | Biological process | negative regulation of DNA-templated transcription |
GO_0033209 | Biological process | tumor necrosis factor-mediated signaling pathway |
GO_0045815 | Biological process | transcription initiation-coupled chromatin remodeling |
GO_0045944 | Biological process | positive regulation of transcription by RNA polymerase II |
GO_2000774 | Biological process | positive regulation of cellular senescence |
GO_0010165 | Biological process | response to X-ray |
GO_0030512 | Biological process | negative regulation of transforming growth factor beta receptor signaling pathway |
GO_0008340 | Biological process | determination of adult lifespan |
GO_0048539 | Biological process | bone marrow development |
GO_0019661 | Biological process | glucose catabolic process to lactate via pyruvate |
GO_0006974 | Biological process | DNA damage response |
GO_0006983 | Biological process | ER overload response |
GO_2000378 | Biological process | negative regulation of reactive oxygen species metabolic process |
GO_0010666 | Biological process | positive regulation of cardiac muscle cell apoptotic process |
GO_0062100 | Biological process | positive regulation of programmed necrotic cell death |
GO_0000122 | Biological process | negative regulation of transcription by RNA polymerase II |
GO_0071494 | Biological process | cellular response to UV-C |
GO_0043525 | Biological process | positive regulation of neuron apoptotic process |
GO_0065003 | Biological process | protein-containing complex assembly |
GO_1990144 | Biological process | intrinsic apoptotic signaling pathway in response to hypoxia |
GO_0072331 | Biological process | signal transduction by p53 class mediator |
GO_0050731 | Biological process | positive regulation of peptidyl-tyrosine phosphorylation |
GO_0051276 | Biological process | chromosome organization |
GO_0010659 | Biological process | cardiac muscle cell apoptotic process |
GO_0090398 | Biological process | cellular senescence |
GO_0048512 | Biological process | circadian behavior |
GO_2000269 | Biological process | regulation of fibroblast apoptotic process |
GO_0008104 | Biological process | protein localization |
GO_0007179 | Biological process | transforming growth factor beta receptor signaling pathway |
GO_0014009 | Biological process | glial cell proliferation |
GO_0030330 | Biological process | DNA damage response, signal transduction by p53 class mediator |
GO_0071456 | Biological process | cellular response to hypoxia |
GO_0016605 | Cellular component | PML body |
GO_0005634 | Cellular component | nucleus |
GO_0005730 | Cellular component | nucleolus |
GO_0043073 | Cellular component | germ cell nucleus |
GO_0005654 | Cellular component | nucleoplasm |
GO_0017053 | Cellular component | transcription repressor complex |
GO_0016604 | Cellular component | nuclear body |
GO_0035861 | Cellular component | site of double-strand break |
GO_0005739 | Cellular component | mitochondrion |
GO_0005813 | Cellular component | centrosome |
GO_0005657 | Cellular component | replication fork |
GO_0032991 | Cellular component | protein-containing complex |
GO_0005783 | Cellular component | endoplasmic reticulum |
GO_0005737 | Cellular component | cytoplasm |
GO_0000785 | Cellular component | chromatin |
GO_0005667 | Cellular component | transcription regulator complex |
GO_0016363 | Cellular component | nuclear matrix |
GO_0005829 | Cellular component | cytosol |
GO_0005759 | Cellular component | mitochondrial matrix |
GO_0140677 | Molecular function | molecular function activator activity |
GO_0001228 | Molecular function | DNA-binding transcription activator activity, RNA polymerase II-specific |
GO_0001046 | Molecular function | core promoter sequence-specific DNA binding |
GO_0042802 | Molecular function | identical protein binding |
GO_0031625 | Molecular function | ubiquitin protein ligase binding |
GO_0008270 | Molecular function | zinc ion binding |
GO_0046982 | Molecular function | protein heterodimerization activity |
GO_0030971 | Molecular function | receptor tyrosine kinase binding |
GO_0035033 | Molecular function | histone deacetylase regulator activity |
GO_0051721 | Molecular function | protein phosphatase 2A binding |
GO_0000978 | Molecular function | RNA polymerase II cis-regulatory region sequence-specific DNA binding |
GO_1990841 | Molecular function | promoter-specific chromatin binding |
GO_0003682 | Molecular function | chromatin binding |
GO_0071889 | Molecular function | 14-3-3 protein binding |
GO_0061629 | Molecular function | RNA polymerase II-specific DNA-binding transcription factor binding |
GO_0140296 | Molecular function | general transcription initiation factor binding |
GO_0003730 | Molecular function | mRNA 3'-UTR binding |
GO_0042826 | Molecular function | histone deacetylase binding |
GO_0019899 | Molecular function | enzyme binding |
GO_0000987 | Molecular function | cis-regulatory region sequence-specific DNA binding |
GO_0000976 | Molecular function | transcription cis-regulatory region binding |
GO_0001094 | Molecular function | TFIID-class transcription factor complex binding |
GO_0002020 | Molecular function | protease binding |
GO_0097371 | Molecular function | MDM2/MDM4 family protein binding |
GO_0051087 | Molecular function | protein-folding chaperone binding |
GO_0003700 | Molecular function | DNA-binding transcription factor activity |
GO_0002039 | Molecular function | p53 binding |
GO_0005507 | Molecular function | copper ion binding |
GO_0005515 | Molecular function | protein binding |
GO_0003677 | Molecular function | DNA binding |
GO_0001227 | Molecular function | DNA-binding transcription repressor activity, RNA polymerase II-specific |
GO_0000981 | Molecular function | DNA-binding transcription factor activity, RNA polymerase II-specific |
GO_0036310 | Molecular function | ATP-dependent DNA/DNA annealing activity |
GO_0097718 | Molecular function | disordered domain specific binding |
Gene name | TP53 |
Protein name | Cellular tumor antigen p53 (Tumor suppressor p53) Cellular tumor antigen p53 (Antigen NY-CO-13) (Phosphoprotein p53) (Tumor suppressor p53) Tumor protein p53 Truncated TP53 p53 Truncated p53 Truncated tumor protein p53 transcript variant 1 p53-Ex10 isoform Cellular tumor antigen p53 Tumor suppressor p53 Truncated p53 splice variant TP53 |
Synonyms | P53 p53 |
Description | FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type . Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process . One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. Its pro-apoptotic activity is activated via its interaction with PPP1R13B/ASPP1 or TP53BP2/ASPP2 . However, this activity is inhibited when the interaction with PPP1R13B/ASPP1 or TP53BP2/ASPP2 is displaced by PPP1R13L/iASPP . In cooperation with mitochondrial PPIF is involved in activating oxidative stress-induced necrosis; the function is largely independent of transcription. Induces the transcription of long intergenic non-coding RNA p21 (lincRNA-p21) and lincRNA-Mkln1. LincRNA-p21 participates in TP53-dependent transcriptional repression leading to apoptosis and seems to have an effect on cell-cycle regulation. Implicated in Notch signaling cross-over. Prevents CDK7 kinase activity when associated to CAK complex in response to DNA damage, thus stopping cell cycle progression. Isoform 2 enhances the transactivation activity of isoform 1 from some but not all TP53-inducible promoters. Isoform 4 suppresses transactivation activity and impairs growth suppression mediated by isoform 1. Isoform 7 inhibits isoform 1-mediated apoptosis. Regulates the circadian clock by repressing CLOCK-BMAL1-mediated transcriptional activation of PER2 . . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . FUNCTION: Acts as a tumor suppressor in many tumor types; induces growth arrest or apoptosis depending on the physiological circumstances and cell type. Involved in cell cycle regulation as a trans-activator that acts to negatively regulate cell division by controlling a set of genes required for this process. One of the activated genes is an inhibitor of cyclin-dependent kinases. Apoptosis induction seems to be mediated either by stimulation of BAX and FAS antigen expression, or by repression of Bcl-2 expression. . |
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