Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
prostate | 100% | 7624.57 | 245 / 245 | 99% | 48.89 | 496 / 502 |
thymus | 100% | 8491.04 | 653 / 653 | 97% | 35.63 | 585 / 605 |
skin | 100% | 6166.67 | 1809 / 1809 | 95% | 43.35 | 449 / 472 |
kidney | 100% | 4988.72 | 89 / 89 | 95% | 27.74 | 855 / 901 |
pancreas | 100% | 4806.22 | 328 / 328 | 92% | 16.83 | 163 / 178 |
breast | 100% | 6667.12 | 459 / 459 | 91% | 82.46 | 1017 / 1118 |
esophagus | 100% | 4400.68 | 1444 / 1445 | 90% | 35.48 | 165 / 183 |
brain | 99% | 3520.88 | 2605 / 2642 | 91% | 22.08 | 645 / 705 |
uterus | 100% | 7391.34 | 170 / 170 | 87% | 23.31 | 401 / 459 |
lung | 100% | 5898.02 | 576 / 578 | 88% | 30.00 | 1011 / 1155 |
ovary | 100% | 9219.19 | 180 / 180 | 85% | 24.22 | 364 / 430 |
adrenal gland | 100% | 6419.53 | 258 / 258 | 84% | 28.31 | 193 / 230 |
intestine | 100% | 5860.39 | 966 / 966 | 83% | 14.23 | 440 / 527 |
bladder | 100% | 5987.24 | 21 / 21 | 79% | 14.81 | 400 / 504 |
stomach | 100% | 4462.98 | 359 / 359 | 79% | 14.57 | 225 / 286 |
blood vessel | 100% | 9923.52 | 1335 / 1335 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 100% | 41.76 | 80 / 80 |
spleen | 100% | 3540.84 | 241 / 241 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 100% | 8.08 | 1 / 1 |
adipose | 100% | 4259.59 | 1203 / 1204 | 0% | 0 | 0 / 0 |
heart | 97% | 3688.51 | 834 / 861 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 91% | 24.48 | 41 / 45 |
muscle | 91% | 1883.62 | 728 / 803 | 0% | 0 | 0 / 0 |
peripheral blood | 76% | 3092.34 | 708 / 929 | 0% | 0 | 0 / 0 |
liver | 1% | 16.21 | 2 / 226 | 10% | 1.55 | 42 / 406 |
lymph node | 0% | 0 | 0 / 0 | 3% | 0.24 | 1 / 29 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
GO_0071560 | Biological process | cellular response to transforming growth factor beta stimulus |
GO_0071260 | Biological process | cellular response to mechanical stimulus |
GO_0048009 | Biological process | insulin-like growth factor receptor signaling pathway |
GO_0007275 | Biological process | multicellular organism development |
GO_0071392 | Biological process | cellular response to estradiol stimulus |
GO_0006955 | Biological process | immune response |
GO_0043066 | Biological process | negative regulation of apoptotic process |
GO_0035094 | Biological process | response to nicotine |
GO_0120162 | Biological process | positive regulation of cold-induced thermogenesis |
GO_0043491 | Biological process | phosphatidylinositol 3-kinase/protein kinase B signal transduction |
GO_0021549 | Biological process | cerebellum development |
GO_0051897 | Biological process | positive regulation of phosphatidylinositol 3-kinase/protein kinase B signal transduction |
GO_0032467 | Biological process | positive regulation of cytokinesis |
GO_0043243 | Biological process | positive regulation of protein-containing complex disassembly |
GO_0034224 | Biological process | cellular response to zinc ion starvation |
GO_1904385 | Biological process | cellular response to angiotensin |
GO_0046328 | Biological process | regulation of JNK cascade |
GO_0071333 | Biological process | cellular response to glucose stimulus |
GO_1990314 | Biological process | cellular response to insulin-like growth factor stimulus |
GO_0071549 | Biological process | cellular response to dexamethasone stimulus |
GO_0007165 | Biological process | signal transduction |
GO_0003230 | Biological process | cardiac atrium development |
GO_0033197 | Biological process | response to vitamin E |
GO_1904045 | Biological process | cellular response to aldosterone |
GO_0048680 | Biological process | positive regulation of axon regeneration |
GO_0097062 | Biological process | dendritic spine maintenance |
GO_0046777 | Biological process | protein autophosphorylation |
GO_1902065 | Biological process | response to L-glutamate |
GO_1904193 | Biological process | negative regulation of cholangiocyte apoptotic process |
GO_0044849 | Biological process | estrous cycle |
GO_0021766 | Biological process | hippocampus development |
GO_0033690 | Biological process | positive regulation of osteoblast proliferation |
GO_0048661 | Biological process | positive regulation of smooth muscle cell proliferation |
GO_0043279 | Biological process | response to alkaloid |
GO_0071394 | Biological process | cellular response to testosterone stimulus |
GO_0045471 | Biological process | response to ethanol |
GO_1903944 | Biological process | negative regulation of hepatocyte apoptotic process |
GO_0097242 | Biological process | amyloid-beta clearance |
GO_0007409 | Biological process | axonogenesis |
GO_0038083 | Biological process | peptidyl-tyrosine autophosphorylation |
GO_0033674 | Biological process | positive regulation of kinase activity |
GO_0008286 | Biological process | insulin receptor signaling pathway |
GO_0045056 | Biological process | transcytosis |
GO_0008284 | Biological process | positive regulation of cell population proliferation |
GO_0051054 | Biological process | positive regulation of DNA metabolic process |
GO_1904646 | Biological process | cellular response to amyloid-beta |
GO_0071393 | Biological process | cellular response to progesterone stimulus |
GO_0030010 | Biological process | establishment of cell polarity |
GO_0043410 | Biological process | positive regulation of MAPK cascade |
GO_0030335 | Biological process | positive regulation of cell migration |
GO_0043409 | Biological process | negative regulation of MAPK cascade |
GO_0090031 | Biological process | positive regulation of steroid hormone biosynthetic process |
GO_0090398 | Biological process | cellular senescence |
GO_0010656 | Biological process | negative regulation of muscle cell apoptotic process |
GO_0005899 | Cellular component | insulin receptor complex |
GO_0043025 | Cellular component | neuronal cell body |
GO_0043231 | Cellular component | intracellular membrane-bounded organelle |
GO_1902911 | Cellular component | protein kinase complex |
GO_0043235 | Cellular component | receptor complex |
GO_0005886 | Cellular component | plasma membrane |
GO_0016020 | Cellular component | membrane |
GO_0030315 | Cellular component | T-tubule |
GO_0035867 | Cellular component | alphav-beta3 integrin-IGF-1-IGF1R complex |
GO_0030424 | Cellular component | axon |
GO_0005901 | Cellular component | caveola |
GO_0043548 | Molecular function | phosphatidylinositol 3-kinase binding |
GO_0005009 | Molecular function | insulin receptor activity |
GO_0001965 | Molecular function | G-protein alpha-subunit binding |
GO_0042802 | Molecular function | identical protein binding |
GO_0005524 | Molecular function | ATP binding |
GO_0005520 | Molecular function | insulin-like growth factor binding |
GO_0043559 | Molecular function | insulin binding |
GO_0005010 | Molecular function | insulin-like growth factor receptor activity |
GO_0004713 | Molecular function | protein tyrosine kinase activity |
GO_0031994 | Molecular function | insulin-like growth factor I binding |
GO_0005515 | Molecular function | protein binding |
GO_0005158 | Molecular function | insulin receptor binding |
GO_0044877 | Molecular function | protein-containing complex binding |
GO_0043560 | Molecular function | insulin receptor substrate binding |
GO_0140318 | Molecular function | protein transporter activity |
Gene name | IGF1R |
Protein name | Insulin like growth factor 1 receptor IGF1R protein receptor protein-tyrosine kinase (EC 2.7.10.1) Insulin-like growth factor 1 receptor (EC 2.7.10.1) (Insulin-like growth factor I receptor) (IGF-I receptor) (CD antigen CD221) [Cleaved into: Insulin-like growth factor 1 receptor alpha chain; Insulin-like growth factor 1 receptor beta chain] Tyrosine-protein kinase receptor (EC 2.7.10.1) |
Synonyms | |
Description | FUNCTION: Receptor tyrosine kinase which mediates actions of insulin-like growth factor 1 (IGF1). Binds IGF1 with high affinity and IGF2 and insulin (INS) with a lower affinity. The activated IGF1R is involved in cell growth and survival control. IGF1R is crucial for tumor transformation and survival of malignant cell. Ligand binding activates the receptor kinase, leading to receptor autophosphorylation, and tyrosines phosphorylation of multiple substrates, that function as signaling adapter proteins including, the insulin-receptor substrates (IRS1/2), Shc and 14-3-3 proteins. Phosphorylation of IRSs proteins lead to the activation of two main signaling pathways: the PI3K-AKT/PKB pathway and the Ras-MAPK pathway. The result of activating the MAPK pathway is increased cellular proliferation, whereas activating the PI3K pathway inhibits apoptosis and stimulates protein synthesis. Phosphorylated IRS1 can activate the 85 kDa regulatory subunit of PI3K (PIK3R1), leading to activation of several downstream substrates, including protein AKT/PKB. AKT phosphorylation, in turn, enhances protein synthesis through mTOR activation and triggers the antiapoptotic effects of IGFIR through phosphorylation and inactivation of BAD. In parallel to PI3K-driven signaling, recruitment of Grb2/SOS by phosphorylated IRS1 or Shc leads to recruitment of Ras and activation of the ras-MAPK pathway. In addition to these two main signaling pathways IGF1R signals also through the Janus kinase/signal transducer and activator of transcription pathway (JAK/STAT). Phosphorylation of JAK proteins can lead to phosphorylation/activation of signal transducers and activators of transcription (STAT) proteins. In particular activation of STAT3, may be essential for the transforming activity of IGF1R. The JAK/STAT pathway activates gene transcription and may be responsible for the transforming activity. JNK kinases can also be activated by the IGF1R. IGF1 exerts inhibiting activities on JNK activation via phosphorylation and inhibition of MAP3K5/ASK1, which is able to directly associate with the IGF1R.; FUNCTION: When present in a hybrid receptor with INSR, binds IGF1. PubMed:12138094 shows that hybrid receptors composed of IGF1R and INSR isoform Long are activated with a high affinity by IGF1, with low affinity by IGF2 and not significantly activated by insulin, and that hybrid receptors composed of IGF1R and INSR isoform Short are activated by IGF1, IGF2 and insulin. In contrast, PubMed:16831875 shows that hybrid receptors composed of IGF1R and INSR isoform Long and hybrid receptors composed of IGF1R and INSR isoform Short have similar binding characteristics, both bind IGF1 and have a low affinity for insulin. |
Accessions | Q3B833 ENST00000558355.1 C9J5X1 P08069 ENST00000558898.1 H0YMJ5 ENST00000649865.1 ENST00000560144.1 ENST00000650285.1 H0YNR0 H3BSZ8 |