Insufficient scRNA-seq data for expression of IRGM at single-cell level.
Insufficient scRNA-seq data for expression of IRGM at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
bladder | 100% | 11.24 | 21 / 21 | 2% | 0.03 | 8 / 504 |
spleen | 99% | 18.82 | 239 / 241 | 0% | 0 | 0 / 0 |
lung | 96% | 12.61 | 557 / 578 | 3% | 0.04 | 30 / 1155 |
adipose | 95% | 12.78 | 1139 / 1204 | 0% | 0 | 0 / 0 |
stomach | 91% | 9.16 | 328 / 359 | 1% | 0.01 | 3 / 286 |
peripheral blood | 92% | 17.00 | 851 / 929 | 0% | 0 | 0 / 0 |
breast | 89% | 9.83 | 410 / 459 | 1% | 0.02 | 9 / 1118 |
intestine | 84% | 10.96 | 815 / 966 | 1% | 0.09 | 4 / 527 |
thymus | 81% | 6.12 | 528 / 653 | 3% | 0.05 | 17 / 605 |
uterus | 79% | 5.59 | 135 / 170 | 3% | 0.09 | 14 / 459 |
ovary | 80% | 5.24 | 144 / 180 | 0% | 0.01 | 2 / 430 |
prostate | 79% | 6.59 | 194 / 245 | 1% | 0.05 | 4 / 502 |
kidney | 75% | 13.73 | 67 / 89 | 1% | 0.01 | 7 / 901 |
esophagus | 72% | 4.40 | 1042 / 1445 | 4% | 0.06 | 7 / 183 |
blood vessel | 74% | 4.83 | 990 / 1335 | 0% | 0 | 0 / 0 |
skin | 61% | 2.79 | 1097 / 1809 | 0% | 0.01 | 2 / 472 |
liver | 51% | 2.58 | 115 / 226 | 3% | 0.07 | 13 / 406 |
adrenal gland | 53% | 2.08 | 136 / 258 | 0% | 0 | 0 / 230 |
heart | 52% | 2.83 | 446 / 861 | 0% | 0 | 0 / 0 |
pancreas | 48% | 1.99 | 159 / 328 | 1% | 0.02 | 2 / 178 |
brain | 39% | 1.46 | 1026 / 2642 | 0% | 0.01 | 3 / 705 |
muscle | 36% | 1.49 | 288 / 803 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 80 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
lymph node | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 29 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 45 |
ureter | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 1 |
GO_0006954 | Biological process | inflammatory response |
GO_0061762 | Biological process | CAMKK-AMPK signaling cascade |
GO_0000045 | Biological process | autophagosome assembly |
GO_0045087 | Biological process | innate immune response |
GO_1900226 | Biological process | negative regulation of NLRP3 inflammasome complex assembly |
GO_0050829 | Biological process | defense response to Gram-negative bacterium |
GO_0033138 | Biological process | positive regulation of peptidyl-serine phosphorylation |
GO_0060335 | Biological process | positive regulation of type II interferon-mediated signaling pathway |
GO_0061635 | Biological process | regulation of protein complex stability |
GO_0071211 | Biological process | protein targeting to vacuole involved in autophagy |
GO_0160049 | Biological process | negative regulation of cGAS/STING signaling pathway |
GO_0001934 | Biological process | positive regulation of protein phosphorylation |
GO_0031648 | Biological process | protein destabilization |
GO_0035458 | Biological process | cellular response to interferon-beta |
GO_0065003 | Biological process | protein-containing complex assembly |
GO_0043032 | Biological process | positive regulation of macrophage activation |
GO_1904417 | Biological process | positive regulation of xenophagy |
GO_0032480 | Biological process | negative regulation of type I interferon production |
GO_0098586 | Biological process | cellular response to virus |
GO_0050728 | Biological process | negative regulation of inflammatory response |
GO_0010508 | Biological process | positive regulation of autophagy |
GO_0050687 | Biological process | negative regulation of defense response to virus |
GO_0050821 | Biological process | protein stabilization |
GO_1905673 | Biological process | positive regulation of lysosome organization |
GO_0042742 | Biological process | defense response to bacterium |
GO_1901526 | Biological process | positive regulation of mitophagy |
GO_0043254 | Biological process | regulation of protein-containing complex assembly |
GO_0071222 | Biological process | cellular response to lipopolysaccharide |
GO_0032689 | Biological process | negative regulation of type II interferon production |
GO_0070431 | Biological process | nucleotide-binding oligomerization domain containing 2 signaling pathway |
GO_1901098 | Biological process | positive regulation of autophagosome maturation |
GO_0010800 | Biological process | positive regulation of peptidyl-threonine phosphorylation |
GO_0097352 | Biological process | autophagosome maturation |
GO_0061739 | Biological process | protein lipidation involved in autophagosome assembly |
GO_0071902 | Biological process | positive regulation of protein serine/threonine kinase activity |
GO_0043124 | Biological process | negative regulation of canonical NF-kappaB signal transduction |
GO_0031902 | Cellular component | late endosome membrane |
GO_0005789 | Cellular component | endoplasmic reticulum membrane |
GO_0005739 | Cellular component | mitochondrion |
GO_0031966 | Cellular component | mitochondrial membrane |
GO_0000421 | Cellular component | autophagosome membrane |
GO_0005794 | Cellular component | Golgi apparatus |
GO_0000139 | Cellular component | Golgi membrane |
GO_0005829 | Cellular component | cytosol |
GO_0001891 | Cellular component | phagocytic cup |
GO_0030670 | Cellular component | phagocytic vesicle membrane |
GO_0042995 | Cellular component | cell projection |
GO_0005765 | Cellular component | lysosomal membrane |
GO_0050700 | Molecular function | CARD domain binding |
GO_0051434 | Molecular function | BH3 domain binding |
GO_0019901 | Molecular function | protein kinase binding |
GO_0003925 | Molecular function | G protein activity |
GO_0043539 | Molecular function | protein serine/threonine kinase activator activity |
GO_0005525 | Molecular function | GTP binding |
GO_0030674 | Molecular function | protein-macromolecule adaptor activity |
GO_0005515 | Molecular function | protein binding |
GO_0003924 | Molecular function | GTPase activity |
Gene name | IRGM |
Protein name | Immunity-related GTPase family M protein (EC 3.6.5.-) (Immunity-related GTPase family M protein 1) (Interferon-inducible protein 1) (LPS-stimulated RAW 264.7 macrophage protein 47 homolog) (LRG-47) Immunity-related GTPase family, M Immunity related GTPase M |
Synonyms | IRGM1 IFI1 LRG47 |
Description | FUNCTION: Immunity-related GTPase that plays important roles in innate immunity and inflammatory response . Acts as a dynamin-like protein that binds to intracellular membranes and promotes remodeling and trafficking of those membranes (By similarity). Required for clearance of acute protozoan and bacterial infections by interacting with autophagy and lysosome regulatory proteins, thereby promoting the fusion of phagosomes with lysosomes for efficient degradation of cargo including microbes . Regulates selective autophagy, including xenophagy and mitophagy, both directly and indirectly . Directly regulates autophagy by acting as a molecular adapter that promotes the coassembly of the core autophagy machinery to mediate antimicrobial defense: IRGM (1) activates AMPK, which in turn phosphorylates ULK1 and BECN1 to induce autophagy, (2) promotes the coassembly of ULK1 and BECN1, enhancing BECN1-interacting partners and (3) influences the composition of the BECN1 complex, by competing with the negative regulators BCL2 and RUBCN, to trigger autophagy . Also activates autophagy by promoting recruitment of STX17 to autophagosomes . In collaboration with ATG8 proteins, regulate lysosomal biogenesis, a fundamental process for any autophagic pathway, by promoting TFEB dephosphorylation . Also modulates autophagy by assisting with autophagosome formation and preventing lysosomal deacidification (By similarity). While activating autophagy, acts as a key negative regulator of the inflammatory and interferon responses both by (1) promoting mitophagy and (2) mediating autophagy-dependent degradation of effectors of the inflammatory response . Promotes degradation of damaged and IFNG/IFN-gamma-stressed mitochondria via mitophagy, preventing cytosolic release of ligands that activate inflammation . Acts as a suppressor of inflammation by promoting recruitment of inflammation effectors, such as CGAS, RIGI/RIG-I and NLRP3, to autophagosome membranes, leading to their SQSTM1/p62-dependent autophagic degradation . Also directly inhibits assembly of the NLRP3 inflammasome by preventing the association between NLRP3 and PYCARD . Acts as a negative regulator of antiviral innate immune response by suppressing the RIPK2-dependent pro-inflammatory response: mediates recruitment of RIPosomes, composed of RIPK2 and NOD1 or NOD2, to autophagosome membranes, promoting their SQSTM1/p62-dependent autophagic degradation . .; FUNCTION: [Isoform IRGMd]: Acts as a positive regulator of mitophagy in response to intracellular mycobacteria infection: specifically binds cardiolipin, leading to its translocation to mitochondria, where it promotes affected mitochondrial fission and mitophagy. .; FUNCTION: (Microbial infection) Following infection by hepatitis C virus (HCV), promotes HCV-triggered membrane remodeling, leading to autophagy and Golgi fragmentation, a step required for HCV replication. . |
Accessions | D9N2U4 ENST00000522154.2 [A1A4Y4-1] A0A9H4B933 A1A4Y4 ENST00000520549.1 |