Name | Number of supported studies | Average coverage | |
---|---|---|---|
endothelial cell | 5 studies | 21% ± 3% | |
endothelial cell of vascular tree | 4 studies | 18% ± 2% |
Insufficient scRNA-seq data for expression of SNAI1 at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
breast | 99% | 1260.00 | 454 / 459 | 95% | 13.45 | 1064 / 1118 |
lung | 99% | 1286.43 | 574 / 578 | 91% | 11.82 | 1055 / 1155 |
thymus | 100% | 1287.63 | 652 / 653 | 81% | 12.16 | 489 / 605 |
intestine | 79% | 315.72 | 762 / 966 | 96% | 14.03 | 507 / 527 |
adrenal gland | 100% | 857.31 | 257 / 258 | 70% | 13.84 | 162 / 230 |
stomach | 71% | 352.81 | 254 / 359 | 99% | 15.61 | 282 / 286 |
bladder | 90% | 435.00 | 19 / 21 | 77% | 9.76 | 387 / 504 |
esophagus | 73% | 301.30 | 1054 / 1445 | 93% | 13.04 | 170 / 183 |
kidney | 93% | 575.84 | 83 / 89 | 69% | 13.12 | 619 / 901 |
skin | 84% | 380.72 | 1514 / 1809 | 73% | 15.34 | 345 / 472 |
uterus | 82% | 400.65 | 140 / 170 | 73% | 10.84 | 335 / 459 |
prostate | 87% | 315.33 | 212 / 245 | 58% | 3.97 | 292 / 502 |
pancreas | 38% | 97.81 | 123 / 328 | 96% | 22.94 | 170 / 178 |
ovary | 44% | 140.91 | 79 / 180 | 80% | 8.46 | 343 / 430 |
liver | 53% | 188.27 | 120 / 226 | 52% | 4.99 | 213 / 406 |
spleen | 100% | 736.59 | 240 / 241 | 0% | 0 | 0 / 0 |
adipose | 100% | 1475.97 | 1198 / 1204 | 0% | 0 | 0 / 0 |
blood vessel | 95% | 890.95 | 1269 / 1335 | 0% | 0 | 0 / 0 |
heart | 80% | 581.59 | 687 / 861 | 0% | 0 | 0 / 0 |
brain | 41% | 109.22 | 1095 / 2642 | 32% | 3.34 | 227 / 705 |
tonsil | 0% | 0 | 0 / 0 | 69% | 4.69 | 31 / 45 |
peripheral blood | 40% | 256.75 | 375 / 929 | 0% | 0 | 0 / 0 |
muscle | 25% | 57.27 | 200 / 803 | 0% | 0 | 0 / 0 |
lymph node | 0% | 0 | 0 / 0 | 24% | 1.86 | 7 / 29 |
eye | 0% | 0 | 0 / 0 | 4% | 0.14 | 3 / 80 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 1 |
GO_0001837 | Biological process | epithelial to mesenchymal transition |
GO_0003198 | Biological process | epithelial to mesenchymal transition involved in endocardial cushion formation |
GO_0007219 | Biological process | Notch signaling pathway |
GO_0060806 | Biological process | negative regulation of cell differentiation involved in embryonic placenta development |
GO_0010631 | Biological process | epithelial cell migration |
GO_0001649 | Biological process | osteoblast differentiation |
GO_0031069 | Biological process | hair follicle morphogenesis |
GO_0003180 | Biological process | aortic valve morphogenesis |
GO_0000122 | Biological process | negative regulation of transcription by RNA polymerase II |
GO_0060536 | Biological process | cartilage morphogenesis |
GO_0060070 | Biological process | canonical Wnt signaling pathway |
GO_0060707 | Biological process | trophoblast giant cell differentiation |
GO_0060972 | Biological process | left/right pattern formation |
GO_0070828 | Biological process | heterochromatin organization |
GO_0060021 | Biological process | roof of mouth development |
GO_0045893 | Biological process | positive regulation of DNA-templated transcription |
GO_0001707 | Biological process | mesoderm formation |
GO_0030335 | Biological process | positive regulation of cell migration |
GO_2000810 | Biological process | regulation of bicellular tight junction assembly |
GO_1902230 | Biological process | negative regulation of intrinsic apoptotic signaling pathway in response to DNA damage |
GO_0006355 | Biological process | regulation of DNA-templated transcription |
GO_0010718 | Biological process | positive regulation of epithelial to mesenchymal transition |
GO_0010957 | Biological process | negative regulation of vitamin D biosynthetic process |
GO_0043518 | Biological process | negative regulation of DNA damage response, signal transduction by p53 class mediator |
GO_0005654 | Cellular component | nucleoplasm |
GO_0001650 | Cellular component | fibrillar center |
GO_0005829 | Cellular component | cytosol |
GO_0005721 | Cellular component | pericentric heterochromatin |
GO_0005737 | Cellular component | cytoplasm |
GO_0043231 | Cellular component | intracellular membrane-bounded organelle |
GO_0005634 | Cellular component | nucleus |
GO_0000978 | Molecular function | RNA polymerase II cis-regulatory region sequence-specific DNA binding |
GO_0000977 | Molecular function | RNA polymerase II transcription regulatory region sequence-specific DNA binding |
GO_0001227 | Molecular function | DNA-binding transcription repressor activity, RNA polymerase II-specific |
GO_0019900 | Molecular function | kinase binding |
GO_1990837 | Molecular function | sequence-specific double-stranded DNA binding |
GO_0046872 | Molecular function | metal ion binding |
GO_0070888 | Molecular function | E-box binding |
GO_0005515 | Molecular function | protein binding |
Gene name | SNAI1 |
Protein name | Zinc finger protein SNAI1 (Protein snail homolog 1) (Protein sna) |
Synonyms | SNAH |
Description | FUNCTION: Involved in induction of the epithelial to mesenchymal transition (EMT), formation and maintenance of embryonic mesoderm, growth arrest, survival and cell migration. Binds to 3 E-boxes of the E-cadherin/CDH1 gene promoter and to the promoters of CLDN7 and KRT8 and, in association with histone demethylase KDM1A which it recruits to the promoters, causes a decrease in dimethylated H3K4 levels and represses transcription . The N-terminal SNAG domain competes with histone H3 for the same binding site on the histone demethylase complex formed by KDM1A and RCOR1, and thereby inhibits demethylation of histone H3 at 'Lys-4' (in vitro) . During EMT, involved with LOXL2 in negatively regulating pericentromeric heterochromatin transcription (By similarity). SNAI1 recruits LOXL2 to pericentromeric regions to oxidize histone H3 and repress transcription which leads to release of heterochromatin component CBX5/HP1A, enabling chromatin reorganization and acquisition of mesenchymal traits (By similarity). Associates with EGR1 and SP1 to mediate tetradecanoyl phorbol acetate (TPA)-induced up-regulation of CDKN2B, possibly by binding to the CDKN2B promoter region 5'-TCACA-3. In addition, may also activate the CDKN2B promoter by itself. . |
Accessions | ENST00000244050.3 O95863 |