Insufficient scRNA-seq data for expression of NLRP6 at single-cell level.
Insufficient scRNA-seq data for expression of NLRP6 at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
kidney | 98% | 184.47 | 87 / 89 | 72% | 4.84 | 646 / 901 |
liver | 99% | 210.50 | 224 / 226 | 55% | 3.13 | 224 / 406 |
lung | 97% | 163.04 | 562 / 578 | 23% | 1.02 | 270 / 1155 |
stomach | 65% | 42.48 | 233 / 359 | 54% | 3.53 | 155 / 286 |
intestine | 71% | 1074.59 | 690 / 966 | 41% | 3.18 | 217 / 527 |
pancreas | 86% | 50.62 | 282 / 328 | 26% | 0.65 | 47 / 178 |
spleen | 100% | 580.61 | 241 / 241 | 0% | 0 | 0 / 0 |
breast | 73% | 47.80 | 333 / 459 | 24% | 0.64 | 272 / 1118 |
peripheral blood | 92% | 2352.06 | 858 / 929 | 0% | 0 | 0 / 0 |
adipose | 77% | 76.05 | 926 / 1204 | 0% | 0 | 0 / 0 |
bladder | 48% | 57.62 | 10 / 21 | 17% | 0.70 | 88 / 504 |
lymph node | 0% | 0 | 0 / 0 | 62% | 2.12 | 18 / 29 |
esophagus | 28% | 13.09 | 401 / 1445 | 33% | 2.99 | 61 / 183 |
thymus | 47% | 28.02 | 309 / 653 | 13% | 0.47 | 81 / 605 |
prostate | 51% | 25.44 | 125 / 245 | 2% | 0.03 | 10 / 502 |
uterus | 36% | 19.24 | 62 / 170 | 15% | 0.32 | 68 / 459 |
skin | 30% | 13.91 | 545 / 1809 | 19% | 0.45 | 92 / 472 |
blood vessel | 49% | 33.60 | 650 / 1335 | 0% | 0 | 0 / 0 |
ovary | 22% | 9.76 | 40 / 180 | 20% | 0.39 | 87 / 430 |
heart | 23% | 10.14 | 198 / 861 | 0% | 0 | 0 / 0 |
adrenal gland | 20% | 7.25 | 52 / 258 | 2% | 0.05 | 4 / 230 |
tonsil | 0% | 0 | 0 / 0 | 18% | 0.35 | 8 / 45 |
brain | 9% | 3.89 | 229 / 2642 | 5% | 0.10 | 35 / 705 |
eye | 0% | 0 | 0 / 0 | 10% | 0.14 | 8 / 80 |
muscle | 4% | 1.32 | 31 / 803 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 1 |
GO_0051260 | Biological process | protein homooligomerization |
GO_0009617 | Biological process | response to bacterium |
GO_0070255 | Biological process | regulation of mucus secretion |
GO_0051607 | Biological process | defense response to virus |
GO_0070373 | Biological process | negative regulation of ERK1 and ERK2 cascade |
GO_0070269 | Biological process | pyroptotic inflammatory response |
GO_0002862 | Biological process | negative regulation of inflammatory response to antigenic stimulus |
GO_0048874 | Biological process | host-mediated regulation of intestinal microbiota composition |
GO_0002526 | Biological process | acute inflammatory response |
GO_0050727 | Biological process | regulation of inflammatory response |
GO_0042060 | Biological process | wound healing |
GO_0007186 | Biological process | G protein-coupled receptor signaling pathway |
GO_0050830 | Biological process | defense response to Gram-positive bacterium |
GO_0010506 | Biological process | regulation of autophagy |
GO_0034122 | Biological process | negative regulation of toll-like receptor signaling pathway |
GO_0032689 | Biological process | negative regulation of type II interferon production |
GO_0070266 | Biological process | necroptotic process |
GO_0050729 | Biological process | positive regulation of inflammatory response |
GO_0140739 | Biological process | NLRP6 inflammasome complex assembly |
GO_0070946 | Biological process | neutrophil-mediated killing of gram-positive bacterium |
GO_2000494 | Biological process | positive regulation of interleukin-18-mediated signaling pathway |
GO_0043124 | Biological process | negative regulation of canonical NF-kappaB signal transduction |
GO_0140374 | Biological process | antiviral innate immune response |
GO_0002438 | Biological process | acute inflammatory response to antigenic stimulus |
GO_0005886 | Cellular component | plasma membrane |
GO_0005829 | Cellular component | cytosol |
GO_0140738 | Cellular component | NLRP6 inflammasome complex |
GO_0005737 | Cellular component | cytoplasm |
GO_0061702 | Cellular component | canonical inflammasome complex |
GO_0043228 | Cellular component | non-membrane-bounded organelle |
GO_0031965 | Cellular component | nuclear membrane |
GO_0038187 | Molecular function | pattern recognition receptor activity |
GO_0042277 | Molecular function | peptide binding |
GO_0035591 | Molecular function | signaling adaptor activity |
GO_0140693 | Molecular function | molecular condensate scaffold activity |
GO_0005000 | Molecular function | vasopressin receptor activity |
GO_0005524 | Molecular function | ATP binding |
GO_0001530 | Molecular function | lipopolysaccharide binding |
GO_0070891 | Molecular function | lipoteichoic acid binding |
GO_0003725 | Molecular function | double-stranded RNA binding |
Gene name | NLRP6 |
Protein name | NACHT, LRR and PYD domains-containing protein 6 (Angiotensin II/vasopressin receptor) (PYRIN-containing APAF1-like protein 5) |
Synonyms | PYPAF5 NALP6 |
Description | FUNCTION: Acts as the sensor component of the NLRP6 inflammasome, which mediates inflammasome activation in response to various pathogen-associated signals, leading to maturation and secretion of IL1B and IL18 . Inflammasomes are supramolecular complexes that assemble in the cytosol in response to pathogens and other damage-associated signals and play critical roles in innate immunity and inflammation . Acts as a recognition receptor (PRR): recognizes and binds specific pathogens and other damage-associated signals, such as lipoteichoic acid (LTA), a cell-wall component of Gram-positive bacteria, or double stranded RNA (dsRNA) . May also recognize and bind lipopolysaccharide (LPS), a major component of the outer membrane of Gram-negative bacteria; however, LPS is probably not a major activator of the NLRP6 inflammasome . Following LTA- or dsRNA-binding, NLRP6 undergoes liquid-liquid phase separation (LLPS), enhancing multivalent interactions, an essential step for the formation of the NLRP6 inflammasome polymeric complex . The NLRP6 inflammasome acts by promoting recruitment of effector pro-inflammatory caspases (CASP1 and/or CASP4) that catalyze maturation and secretion of IL1B and IL18 in the extracellular milieu . The NLRP6 inflammasome plays a central role in the maintenance of epithelial integrity and host defense against microbial infections in the intestine . Required to restrict infection against Gram-positive bacteria by recognizing lipoteichoic acid (LTA), leading to recruitment of CASP4 and CASP1, and subsequent maturation and secretion of IL1B and IL18 . Involved in intestinal antiviral innate immunity together with DHX15: recognizes and binds viral dsRNA to restrict infection by enteric viruses through the interferon pathway and GSDMD-dependent release of IL18 . Required to prevent infection by the apicomplexan parasite Cryptosporidium in enterocytes by promoting GSDMD-dependent release of IL18 (By similarity). The NLRP6 inflammasome may also regulate the gut microbiota composition by acting as a sensor of microbiota-associated metabolites to form a PYCARD/ASC-dependent inflammasome for downstream IL18 release and secretion of antimicrobial peptides (By similarity). Essential for gut mucosal self-renewal and proliferation (By similarity). Regulate mucus secretion in an inflammasome- and autophagy-dependent manner to prevent invasion by enteric bacteria, (By similarity). During systemic bacterial infections, the NLRP6 inflammasome negatively regulates neutrophil recruitment and neutrophil extracellular traps (NETs) formation (By similarity). May promote peripheral nerve recovery following injury via an inflammasome-independent mechanism (By similarity). . |
Accessions | ENST00000312165.5 [P59044-1] ENST00000534750.6 [P59044-2] P59044 |