Name | Number of supported studies | Average coverage | |
---|---|---|---|
epithelial cell | 3 studies | 29% ± 16% | |
GABAergic neuron | 3 studies | 36% ± 6% | |
glutamatergic neuron | 3 studies | 45% ± 2% | |
astrocyte | 3 studies | 30% ± 7% | |
microglial cell | 3 studies | 20% ± 4% |
Name | Number of supported studies | Average coverage | |
---|---|---|---|
brain | 4 studies | 33% ± 7% |
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
esophagus | 100% | 1127.33 | 1445 / 1445 | 100% | 4.98 | 183 / 183 |
breast | 100% | 1115.93 | 459 / 459 | 99% | 5.30 | 1112 / 1118 |
prostate | 100% | 1302.76 | 245 / 245 | 99% | 4.52 | 499 / 502 |
lung | 99% | 912.58 | 575 / 578 | 100% | 4.08 | 1154 / 1155 |
thymus | 100% | 1562.83 | 653 / 653 | 99% | 5.20 | 600 / 605 |
bladder | 100% | 1119.67 | 21 / 21 | 99% | 3.72 | 499 / 504 |
uterus | 100% | 1246.32 | 170 / 170 | 99% | 3.88 | 454 / 459 |
ovary | 100% | 1312.03 | 180 / 180 | 99% | 3.27 | 424 / 430 |
stomach | 100% | 757.60 | 359 / 359 | 99% | 3.24 | 282 / 286 |
intestine | 100% | 1048.12 | 966 / 966 | 98% | 2.98 | 519 / 527 |
pancreas | 99% | 707.66 | 326 / 328 | 99% | 3.07 | 176 / 178 |
brain | 99% | 766.85 | 2603 / 2642 | 98% | 3.20 | 694 / 705 |
kidney | 100% | 795.27 | 89 / 89 | 92% | 2.86 | 833 / 901 |
skin | 100% | 2067.51 | 1809 / 1809 | 89% | 2.40 | 419 / 472 |
adrenal gland | 100% | 839.32 | 258 / 258 | 85% | 1.96 | 195 / 230 |
liver | 100% | 624.34 | 226 / 226 | 67% | 1.31 | 272 / 406 |
adipose | 100% | 1138.25 | 1204 / 1204 | 0% | 0 | 0 / 0 |
lymph node | 0% | 0 | 0 / 0 | 100% | 2.67 | 29 / 29 |
spleen | 100% | 1147.13 | 241 / 241 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 100% | 4.03 | 45 / 45 |
ureter | 0% | 0 | 0 / 0 | 100% | 1.22 | 1 / 1 |
blood vessel | 100% | 1598.90 | 1334 / 1335 | 0% | 0 | 0 / 0 |
muscle | 100% | 792.72 | 799 / 803 | 0% | 0 | 0 / 0 |
heart | 97% | 818.60 | 831 / 861 | 0% | 0 | 0 / 0 |
peripheral blood | 76% | 632.12 | 709 / 929 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 65% | 1.25 | 52 / 80 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
GO_0006366 | Biological process | transcription by RNA polymerase II |
GO_0007254 | Biological process | JNK cascade |
GO_0033141 | Biological process | positive regulation of peptidyl-serine phosphorylation of STAT protein |
GO_0000012 | Biological process | single strand break repair |
GO_0032786 | Biological process | positive regulation of DNA-templated transcription, elongation |
GO_0010165 | Biological process | response to X-ray |
GO_0000303 | Biological process | response to superoxide |
GO_0010332 | Biological process | response to gamma radiation |
GO_0031175 | Biological process | neuron projection development |
GO_0045739 | Biological process | positive regulation of DNA repair |
GO_2001033 | Biological process | negative regulation of double-strand break repair via nonhomologous end joining |
GO_0045494 | Biological process | photoreceptor cell maintenance |
GO_0006362 | Biological process | transcription elongation by RNA polymerase I |
GO_0010628 | Biological process | positive regulation of gene expression |
GO_0034243 | Biological process | regulation of transcription elongation by RNA polymerase II |
GO_0035264 | Biological process | multicellular organism growth |
GO_0006290 | Biological process | pyrimidine dimer repair |
GO_0032784 | Biological process | regulation of DNA-templated transcription elongation |
GO_0006338 | Biological process | chromatin remodeling |
GO_0097680 | Biological process | double-strand break repair via classical nonhomologous end joining |
GO_0010224 | Biological process | response to UV-B |
GO_0030182 | Biological process | neuron differentiation |
GO_0060261 | Biological process | positive regulation of transcription initiation by RNA polymerase II |
GO_0022008 | Biological process | neurogenesis |
GO_0045945 | Biological process | positive regulation of transcription by RNA polymerase III |
GO_0042262 | Biological process | DNA protection |
GO_0045943 | Biological process | positive regulation of transcription by RNA polymerase I |
GO_0008630 | Biological process | intrinsic apoptotic signaling pathway in response to DNA damage |
GO_1905168 | Biological process | positive regulation of double-strand break repair via homologous recombination |
GO_0009636 | Biological process | response to toxic substance |
GO_0006284 | Biological process | base-excision repair |
GO_0006979 | Biological process | response to oxidative stress |
GO_0002230 | Biological process | positive regulation of defense response to virus by host |
GO_0006283 | Biological process | transcription-coupled nucleotide-excision repair |
GO_0045944 | Biological process | positive regulation of transcription by RNA polymerase II |
GO_0000077 | Biological process | DNA damage checkpoint signaling |
GO_0005730 | Cellular component | nucleolus |
GO_0008023 | Cellular component | transcription elongation factor complex |
GO_0110016 | Cellular component | B-WICH complex |
GO_0005654 | Cellular component | nucleoplasm |
GO_0090734 | Cellular component | site of DNA damage |
GO_0016604 | Cellular component | nuclear body |
GO_0005634 | Cellular component | nucleus |
GO_0003677 | Molecular function | DNA binding |
GO_0043565 | Molecular function | sequence-specific DNA binding |
GO_0003682 | Molecular function | chromatin binding |
GO_0030296 | Molecular function | protein tyrosine kinase activator activity |
GO_0003678 | Molecular function | DNA helicase activity |
GO_0005524 | Molecular function | ATP binding |
GO_0140658 | Molecular function | ATP-dependent chromatin remodeler activity |
GO_0008094 | Molecular function | ATP-dependent activity, acting on DNA |
GO_0005515 | Molecular function | protein binding |
GO_0016887 | Molecular function | ATP hydrolysis activity |
Gene name | ERCC6 |
Protein name | Excision repair cross-complementing rodent repair deficiency, complementation group 6 Chimeric ERCC6-PGBD3 protein (Chimeric CSB-PGBD3 protein) DNA excision repair protein ERCC-6 (EC 3.6.4.-) (ATP-dependent helicase ERCC6) (Cockayne syndrome protein CSB) ERCC excision repair 6, chromatin remodeling factor |
Synonyms | CSB |
Description | FUNCTION: Involved in repair of DNA damage following UV irradiation, acting either in the absence of ERCC6 or synergistically with ERCC6. Involved in the regulation of gene expression. In the absence of ERCC6, induces the expression of genes characteristic of interferon-like antiviral responses. This response is almost completely suppressed in the presence of ERCC6. In the presence of ERCC6, regulates the expression of genes involved in metabolism regulation, including IGFBP5 and IGFBP7. In vitro binds to PGBD3-related transposable elements, called MER85s; these non-autonomous 140 bp elements are characterized by the presence of PGBD3 terminal inverted repeats and the absence of internal transposase ORF. . FUNCTION: Essential factor involved in transcription-coupled nucleotide excision repair which allows RNA polymerase II-blocking lesions to be rapidly removed from the transcribed strand of active genes . Upon DNA-binding, it locally modifies DNA conformation by wrapping the DNA around itself, thereby modifying the interface between stalled RNA polymerase II and DNA . It is required for transcription-coupled repair complex formation . It recruits the CSA complex (DCX(ERCC8) complex), nucleotide excision repair proteins and EP300 to the sites of RNA polymerase II-blocking lesions . Plays an important role in regulating the choice of the DNA double-strand breaks (DSBs) repair pathway and G2/M checkpoint activation; DNA-dependent ATPase activity is essential for this function . Regulates the DNA repair pathway choice by inhibiting non-homologous end joining (NHEJ), thereby promoting the homologous recombination (HR)-mediated repair of DSBs during the S/G2 phases of the cell cycle . Mediates the activation of the ATM- and CHEK2-dependent DNA damage responses thus preventing premature entry of cells into mitosis following the induction of DNA DSBs . Acts as a chromatin remodeler at DSBs; DNA-dependent ATPase-dependent activity is essential for this function. Remodels chromatin by evicting histones from chromatin flanking DSBs, limiting RIF1 accumulation at DSBs thereby promoting BRCA1-mediated HR . Required for stable recruitment of ELOA and CUL5 to DNA damage sites . Involved in UV-induced translocation of ERCC8 to the nuclear matrix . Essential for neuronal differentiation and neuritogenesis; regulates transcription and chromatin remodeling activities required during neurogenesis . . |
Accessions | ENST00000680107.1 ENST00000624341.3 ENST00000355832.10 [Q03468-1] ENST00000515869.1 [P0DP91-1] ENST00000681659.1 P0DP91 ENST00000679596.1 A0A7P0TA32 ENST00000462247.1 ENST00000447839.7 [P0DP91-1] Q03468 A0A7P0T7Y4 A0A096LNQ7 A0JP10 D6R9X7 A0A7P0T9G4 |