Insufficient scRNA-seq data for expression of C4B at single-cell level.
Insufficient scRNA-seq data for expression of C4B at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
liver | 100% | 11591.62 | 226 / 226 | 95% | 90.64 | 386 / 406 |
thymus | 100% | 2838.94 | 652 / 653 | 90% | 37.35 | 546 / 605 |
kidney | 88% | 1981.28 | 78 / 89 | 81% | 32.68 | 730 / 901 |
ovary | 99% | 2894.03 | 179 / 180 | 60% | 16.10 | 260 / 430 |
breast | 76% | 926.28 | 351 / 459 | 76% | 41.03 | 848 / 1118 |
adrenal gland | 100% | 11471.88 | 258 / 258 | 28% | 11.08 | 65 / 230 |
lung | 86% | 746.17 | 497 / 578 | 36% | 5.75 | 412 / 1155 |
pancreas | 68% | 865.87 | 222 / 328 | 54% | 7.06 | 96 / 178 |
prostate | 62% | 536.81 | 151 / 245 | 56% | 10.78 | 280 / 502 |
brain | 43% | 275.84 | 1124 / 2642 | 67% | 14.71 | 475 / 705 |
bladder | 95% | 905.95 | 20 / 21 | 12% | 1.61 | 60 / 504 |
intestine | 88% | 1063.48 | 847 / 966 | 13% | 1.98 | 69 / 527 |
spleen | 98% | 969.55 | 235 / 241 | 0% | 0 | 0 / 0 |
uterus | 80% | 772.06 | 136 / 170 | 17% | 3.34 | 78 / 459 |
esophagus | 58% | 714.61 | 844 / 1445 | 30% | 5.41 | 54 / 183 |
stomach | 63% | 428.98 | 227 / 359 | 23% | 3.73 | 66 / 286 |
blood vessel | 74% | 633.71 | 989 / 1335 | 0% | 0 | 0 / 0 |
heart | 68% | 635.57 | 585 / 861 | 0% | 0 | 0 / 0 |
adipose | 56% | 790.30 | 678 / 1204 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 25% | 3.10 | 20 / 80 |
skin | 3% | 10.41 | 50 / 1809 | 8% | 1.14 | 36 / 472 |
lymph node | 0% | 0 | 0 / 0 | 10% | 0.93 | 3 / 29 |
tonsil | 0% | 0 | 0 / 0 | 4% | 0.29 | 2 / 45 |
muscle | 2% | 10.80 | 19 / 803 | 0% | 0 | 0 / 0 |
peripheral blood | 0% | 1.33 | 3 / 929 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 1 |
GO_0006954 | Biological process | inflammatory response |
GO_0045087 | Biological process | innate immune response |
GO_0006956 | Biological process | complement activation |
GO_0006958 | Biological process | complement activation, classical pathway |
GO_2000427 | Biological process | positive regulation of apoptotic cell clearance |
GO_0032490 | Biological process | detection of molecule of bacterial origin |
GO_0008228 | Biological process | opsonization |
GO_0005615 | Cellular component | extracellular space |
GO_0005576 | Cellular component | extracellular region |
GO_0005886 | Cellular component | plasma membrane |
GO_0070062 | Cellular component | extracellular exosome |
GO_0030425 | Cellular component | dendrite |
GO_0072562 | Cellular component | blood microparticle |
GO_0045202 | Cellular component | synapse |
GO_0106139 | Cellular component | symbiont cell surface |
GO_0030424 | Cellular component | axon |
GO_0004866 | Molecular function | endopeptidase inhibitor activity |
GO_0030246 | Molecular function | carbohydrate binding |
GO_0001848 | Molecular function | complement binding |
Gene name | C4B |
Protein name | Complement C4B1a (Complement C4B2) (Complement C4B5) C4B1 (C4B2) (C4B3) (C4B5) Complement protein C4B frameshift mutant Complement C4-B (Complement C4B (Chido blood group)) Complement C4-B Complement component 4B Complement C4B (Chido blood group) Complement C4-B (Basic complement C4) (C3 and PZP-like alpha-2-macroglobulin domain-containing protein 3) [Cleaved into: Complement C4 beta chain; Complement C4-B alpha chain; C4a anaphylatoxin; C4b-B; C4d-B; Complement C4 gamma chain] C4B1 C4B C4B2 (C4B5) |
Synonyms | CPAMD3; CO4 C4B_2 |
Description | FUNCTION: Non-enzymatic component of the C3 and C5 convertases and thus essential for the propagation of the classical complement pathway. Covalently binds to immunoglobulins and immune complexes and enhances the solubilization of immune aggregates and the clearance of IC through CR1 on erythrocytes. C4A isotype is responsible for effective binding to form amide bonds with immune aggregates or protein antigens, while C4B isotype catalyzes the transacylation of the thioester carbonyl group to form ester bonds with carbohydrate antigens.; FUNCTION: Derived from proteolytic degradation of complement C4, C4a anaphylatoxin is a mediator of local inflammatory process. It induces the contraction of smooth muscle, increases vascular permeability and causes histamine release from mast cells and basophilic leukocytes. |
Accessions | Q6U2L6 F5GXS0 A0A140TA29 Q9UMV1 P0C0L5 Q6U2M2 ENST00000375177.9 ENST00000425700.3 ENST00000649658.1 ENST00000552464.2 ENST00000435363.7 ENST00000411583.6 Q6U2H4 A0A3B3ISA6 ENST00000546399.2 Q6U2E7 ENST00000548301.2 ENST00000435500.6 Q6U2E0 ENST00000449788.6 ENST00000647698.1 ENST00000548530.2 Q9UNU2 A0A0G2JL54 A0A3B3ISD7 R9QA77 |