Insufficient scRNA-seq data for expression of SUCNR1 at single-cell level.
Insufficient scRNA-seq data for expression of SUCNR1 at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
thymus | 100% | 2093.92 | 653 / 653 | 80% | 4.50 | 483 / 605 |
kidney | 100% | 1054.13 | 89 / 89 | 76% | 12.77 | 681 / 901 |
lung | 94% | 145.92 | 544 / 578 | 78% | 6.16 | 896 / 1155 |
liver | 92% | 161.32 | 208 / 226 | 62% | 8.07 | 250 / 406 |
breast | 80% | 202.17 | 365 / 459 | 65% | 2.44 | 723 / 1118 |
adrenal gland | 88% | 165.23 | 226 / 258 | 29% | 6.87 | 66 / 230 |
skin | 51% | 36.58 | 917 / 1809 | 50% | 1.81 | 238 / 472 |
spleen | 98% | 573.42 | 235 / 241 | 0% | 0 | 0 / 0 |
adipose | 96% | 368.66 | 1159 / 1204 | 0% | 0 | 0 / 0 |
pancreas | 4% | 1.67 | 12 / 328 | 72% | 5.52 | 128 / 178 |
intestine | 29% | 17.41 | 277 / 966 | 46% | 1.91 | 240 / 527 |
stomach | 9% | 9.30 | 34 / 359 | 64% | 5.72 | 184 / 286 |
esophagus | 15% | 14.31 | 213 / 1445 | 58% | 2.49 | 107 / 183 |
lymph node | 0% | 0 | 0 / 0 | 72% | 3.63 | 21 / 29 |
bladder | 29% | 12.86 | 6 / 21 | 39% | 1.62 | 196 / 504 |
peripheral blood | 67% | 117.78 | 624 / 929 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 60% | 2.19 | 27 / 45 |
blood vessel | 56% | 80.76 | 754 / 1335 | 0% | 0 | 0 / 0 |
ovary | 3% | 1.35 | 5 / 180 | 47% | 1.65 | 204 / 430 |
uterus | 2% | 0.80 | 3 / 170 | 40% | 2.33 | 182 / 459 |
brain | 13% | 8.23 | 332 / 2642 | 24% | 0.52 | 168 / 705 |
heart | 31% | 36.95 | 266 / 861 | 0% | 0 | 0 / 0 |
prostate | 5% | 2.92 | 13 / 245 | 13% | 2.59 | 65 / 502 |
eye | 0% | 0 | 0 / 0 | 6% | 0.10 | 5 / 80 |
muscle | 1% | 0.45 | 8 / 803 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
ureter | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 1 |
GO_0097009 | Biological process | energy homeostasis |
GO_0002281 | Biological process | macrophage activation involved in immune response |
GO_0051592 | Biological process | response to calcium ion |
GO_0050729 | Biological process | positive regulation of inflammatory response |
GO_0007186 | Biological process | G protein-coupled receptor signaling pathway |
GO_0060177 | Biological process | regulation of angiotensin metabolic process |
GO_0002001 | Biological process | renin secretion into blood stream |
GO_0042593 | Biological process | glucose homeostasis |
GO_0050921 | Biological process | positive regulation of chemotaxis |
GO_0070062 | Cellular component | extracellular exosome |
GO_0005886 | Cellular component | plasma membrane |
GO_0038023 | Molecular function | signaling receptor activity |
GO_0004930 | Molecular function | G protein-coupled receptor activity |
GO_0005515 | Molecular function | protein binding |
Gene name | SUCNR1 |
Protein name | Succinate receptor 1 (G-protein coupled receptor 91) (P2Y purinoceptor 1-like) |
Synonyms | GPR91 |
Description | FUNCTION: G protein-coupled receptor for succinate able to mediate signaling through Gq/GNAQ or Gi/GNAI second messengers depending on the cell type and the processes regulated (By similarity) . Succinate-SUCNR1 signaling serves as a link between metabolic stress, inflammation and energy homeostasis . In macrophages, plays a range of immune-regulatory roles. During inflammation, succinate-SUCNR1 signaling may act as an anti-inflammatory mediator or boost inflammation depending on the inflammatory status of cells (By similarity). Hyperpolarizes M2 macrophages versus M1 phenotype through Gq signaling by regulating the transcription of genes involoved in immune function . In activated M1 macrophages, plays a pro-inflammatory role in response to LPS (By similarity). Expressed in dendritic cells, where it is involved in the sensing of immunological danger and enhances immunity. Mediates succinate triggered intracelleular calcium mobilization, induces migratory responses and acts in synergy with Toll-like receptor ligands for the production of proinflammatory cytokines as well as an enhancement of antigen-specific activation of helper T cells . In the small intestine, mediates the activation of tuft cells by dietary succinate and triggers type 2 immunity (By similarity). In adipocytes, plays an important role in the control of energy metabolism. In response to succinate, controls leptin expression in an AMPK-JNK-CEBPA-dependent as well as circadian clock-regulated manner (By similarity). In muscle tissue, is expressed in non-muscle cells and coordinates muscle remodeling in response to the succinate produced during exercise training in a paracrine manner (By similarity). In retina, acts as a mediator of vessel growth during retinal development. In response to succinate, regulates the production of angiogenic factors, including VEGF, by retinal ganglion neurons (By similarity). . |
Accessions | Q9BXA5 ENST00000362032.6 |