Name | Number of supported studies | Average coverage | |
---|---|---|---|
endothelial cell | 7 studies | 19% ± 4% | |
epithelial cell | 4 studies | 35% ± 17% | |
oligodendrocyte | 4 studies | 18% ± 1% | |
fibroblast | 3 studies | 19% ± 3% | |
ciliated cell | 3 studies | 30% ± 16% | |
GABAergic neuron | 3 studies | 31% ± 8% | |
glutamatergic neuron | 3 studies | 40% ± 11% | |
basal cell | 3 studies | 23% ± 5% | |
astrocyte | 3 studies | 23% ± 7% |
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
breast | 100% | 1008.31 | 459 / 459 | 100% | 11.59 | 1118 / 1118 |
esophagus | 100% | 1327.37 | 1445 / 1445 | 100% | 8.60 | 183 / 183 |
lung | 100% | 1128.42 | 578 / 578 | 100% | 11.16 | 1155 / 1155 |
ovary | 100% | 1345.12 | 180 / 180 | 100% | 9.38 | 430 / 430 |
prostate | 100% | 1226.21 | 245 / 245 | 100% | 9.04 | 502 / 502 |
uterus | 100% | 1298.57 | 170 / 170 | 100% | 14.99 | 459 / 459 |
brain | 100% | 1498.32 | 2641 / 2642 | 100% | 13.25 | 705 / 705 |
thymus | 100% | 957.03 | 653 / 653 | 100% | 8.53 | 604 / 605 |
bladder | 100% | 1156.71 | 21 / 21 | 100% | 11.96 | 503 / 504 |
intestine | 100% | 1083.07 | 966 / 966 | 100% | 10.77 | 525 / 527 |
skin | 100% | 1393.90 | 1809 / 1809 | 100% | 13.88 | 470 / 472 |
stomach | 100% | 793.45 | 359 / 359 | 99% | 9.30 | 284 / 286 |
kidney | 100% | 1055.24 | 89 / 89 | 99% | 6.96 | 890 / 901 |
adrenal gland | 100% | 1369.41 | 258 / 258 | 99% | 10.69 | 227 / 230 |
liver | 100% | 514.75 | 226 / 226 | 98% | 6.79 | 399 / 406 |
pancreas | 98% | 501.77 | 321 / 328 | 99% | 9.32 | 177 / 178 |
adipose | 100% | 952.97 | 1204 / 1204 | 0% | 0 | 0 / 0 |
lymph node | 0% | 0 | 0 / 0 | 100% | 11.70 | 29 / 29 |
spleen | 100% | 925.12 | 241 / 241 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 100% | 11.93 | 45 / 45 |
ureter | 0% | 0 | 0 / 0 | 100% | 10.36 | 1 / 1 |
muscle | 100% | 907.36 | 802 / 803 | 0% | 0 | 0 / 0 |
blood vessel | 100% | 1141.62 | 1333 / 1335 | 0% | 0 | 0 / 0 |
heart | 98% | 985.20 | 845 / 861 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 96% | 8.25 | 77 / 80 |
peripheral blood | 73% | 560.35 | 680 / 929 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
GO_0006303 | Biological process | double-strand break repair via nonhomologous end joining |
GO_0036297 | Biological process | interstrand cross-link repair |
GO_0010212 | Biological process | response to ionizing radiation |
GO_0140861 | Biological process | DNA repair-dependent chromatin remodeling |
GO_0045739 | Biological process | positive regulation of DNA repair |
GO_0006302 | Biological process | double-strand break repair |
GO_0006974 | Biological process | DNA damage response |
GO_0006511 | Biological process | ubiquitin-dependent protein catabolic process |
GO_0051865 | Biological process | protein autoubiquitination |
GO_0051301 | Biological process | cell division |
GO_0034244 | Biological process | negative regulation of transcription elongation by RNA polymerase II |
GO_0070936 | Biological process | protein K48-linked ubiquitination |
GO_0070534 | Biological process | protein K63-linked ubiquitination |
GO_1905168 | Biological process | positive regulation of double-strand break repair via homologous recombination |
GO_0035092 | Biological process | sperm DNA condensation |
GO_0045190 | Biological process | isotype switching |
GO_0040029 | Biological process | epigenetic regulation of gene expression |
GO_0042770 | Biological process | signal transduction in response to DNA damage |
GO_0085020 | Biological process | protein K6-linked ubiquitination |
GO_0000151 | Cellular component | ubiquitin ligase complex |
GO_0030496 | Cellular component | midbody |
GO_0005654 | Cellular component | nucleoplasm |
GO_0005829 | Cellular component | cytosol |
GO_0000781 | Cellular component | chromosome, telomeric region |
GO_0035861 | Cellular component | site of double-strand break |
GO_0005634 | Cellular component | nucleus |
GO_0061630 | Molecular function | ubiquitin protein ligase activity |
GO_0042803 | Molecular function | protein homodimerization activity |
GO_0042393 | Molecular function | histone binding |
GO_0003682 | Molecular function | chromatin binding |
GO_0042802 | Molecular function | identical protein binding |
GO_0031625 | Molecular function | ubiquitin protein ligase binding |
GO_0008270 | Molecular function | zinc ion binding |
GO_0005515 | Molecular function | protein binding |
GO_0043130 | Molecular function | ubiquitin binding |
Gene name | RNF8 |
Protein name | E3 ubiquitin-protein ligase RNF8 (hRNF8) (EC 2.3.2.27) (RING finger protein 8) (RING-type E3 ubiquitin transferase RNF8) Alternative protein RNF8 Ring finger protein 8 |
Synonyms | KIAA0646 |
Description | FUNCTION: E3 ubiquitin-protein ligase that plays a key role in DNA damage signaling via 2 distinct roles: by mediating the 'Lys-63'-linked ubiquitination of histones H2A and H2AX and promoting the recruitment of DNA repair proteins at double-strand breaks (DSBs) sites, and by catalyzing 'Lys-48'-linked ubiquitination to remove target proteins from DNA damage sites. Following DNA DSBs, it is recruited to the sites of damage by ATM-phosphorylated MDC1 and catalyzes the 'Lys-63'-linked ubiquitination of histones H2A and H2AX, thereby promoting the formation of TP53BP1 and BRCA1 ionizing radiation-induced foci (IRIF). Also controls the recruitment of UIMC1-BRCC3 (RAP80-BRCC36) and PAXIP1/PTIP to DNA damage sites. Also recruited at DNA interstrand cross-links (ICLs) sites and catalyzes 'Lys-63'-linked ubiquitination of histones H2A and H2AX, leading to recruitment of FAAP20/C1orf86 and Fanconi anemia (FA) complex, followed by interstrand cross-link repair. H2A ubiquitination also mediates the ATM-dependent transcriptional silencing at regions flanking DSBs in cis, a mechanism to avoid collision between transcription and repair intermediates. Promotes the formation of 'Lys-63'-linked polyubiquitin chains via interactions with the specific ubiquitin-conjugating UBE2N/UBC13 and ubiquitinates non-histone substrates such as PCNA. Substrates that are polyubiquitinated at 'Lys-63' are usually not targeted for degradation. Also catalyzes the formation of 'Lys-48'-linked polyubiquitin chains via interaction with the ubiquitin-conjugating UBE2L6/UBCH8, leading to degradation of substrate proteins such as CHEK2, JMJD2A/KDM4A and KU80/XRCC5: it is still unclear how the preference toward 'Lys-48'- versus 'Lys-63'-linked ubiquitination is regulated but it could be due to RNF8 ability to interact with specific E2 specific ligases. For instance, interaction with phosphorylated HERC2 promotes the association between RNF8 and UBE2N/UBC13 and favors the specific formation of 'Lys-63'-linked ubiquitin chains. Promotes non-homologous end joining (NHEJ) by promoting the 'Lys-48'-linked ubiquitination and degradation the of KU80/XRCC5. Following DNA damage, mediates the ubiquitination and degradation of JMJD2A/KDM4A in collaboration with RNF168, leading to unmask H4K20me2 mark and promote the recruitment of TP53BP1 at DNA damage sites . Following DNA damage, mediates the ubiquitination and degradation of POLD4/p12, a subunit of DNA polymerase delta. In the absence of POLD4, DNA polymerase delta complex exhibits higher proofreading activity . In addition to its function in damage signaling, also plays a role in higher-order chromatin structure by mediating extensive chromatin decondensation. Involved in the activation of ATM by promoting histone H2B ubiquitination, which indirectly triggers histone H4 'Lys-16' acetylation (H4K16ac), establishing a chromatin environment that promotes efficient activation of ATM kinase. Required in the testis, where it plays a role in the replacement of histones during spermatogenesis. At uncapped telomeres, promotes the joining of deprotected chromosome ends by inducing H2A ubiquitination and TP53BP1 recruitment, suggesting that it may enhance cancer development by aggravating telomere-induced genome instability in case of telomeric crisis. Promotes the assembly of RAD51 at DNA DSBs in the absence of BRCA1 and TP53BP1 Also involved in class switch recombination in immune system, via its role in regulation of DSBs repair. May be required for proper exit from mitosis after spindle checkpoint activation and may regulate cytokinesis. May play a role in the regulation of RXRA-mediated transcriptional activity. Not involved in RXRA ubiquitination by UBE2E2 . . |
Accessions | H7C4L7 ENST00000373479.9 [O76064-1] ENST00000498460.1 F8WDH8 F8WEW6 C9J858 ENST00000469316.5 ENST00000487950.1 L0R6N6 ENST00000229866.10 [O76064-2] ENST00000469731.5 [O76064-3] ENST00000494320.5 O76064 |