Insufficient scRNA-seq data for expression of TREX1 at single-cell level.
Insufficient scRNA-seq data for expression of TREX1 at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
adrenal gland | 100% | 1200.45 | 258 / 258 | 0% | 0 | 0 / 230 |
bladder | 100% | 988.19 | 21 / 21 | 0% | 0 | 0 / 504 |
breast | 100% | 972.51 | 459 / 459 | 0% | 0 | 0 / 1118 |
esophagus | 100% | 1052.36 | 1445 / 1445 | 0% | 0 | 0 / 183 |
intestine | 100% | 914.13 | 966 / 966 | 0% | 0 | 0 / 527 |
kidney | 100% | 969.01 | 89 / 89 | 0% | 0 | 0 / 901 |
lung | 100% | 1186.34 | 578 / 578 | 0% | 0 | 0 / 1155 |
prostate | 100% | 1475.08 | 245 / 245 | 0% | 0 | 0 / 502 |
skin | 100% | 1719.03 | 1809 / 1809 | 0% | 0 | 0 / 472 |
spleen | 100% | 2167.61 | 241 / 241 | 0% | 0 | 0 / 0 |
stomach | 100% | 822.64 | 359 / 359 | 0% | 0 | 0 / 286 |
thymus | 100% | 1044.79 | 653 / 653 | 0% | 0 | 0 / 605 |
uterus | 100% | 820.28 | 170 / 170 | 0% | 0 | 0 / 459 |
adipose | 100% | 910.67 | 1202 / 1204 | 0% | 0 | 0 / 0 |
brain | 100% | 894.67 | 2633 / 2642 | 0% | 0 | 0 / 705 |
blood vessel | 99% | 785.29 | 1328 / 1335 | 0% | 0 | 0 / 0 |
ovary | 99% | 624.94 | 178 / 180 | 0% | 0 | 0 / 430 |
peripheral blood | 98% | 1669.31 | 911 / 929 | 0% | 0 | 0 / 0 |
pancreas | 98% | 628.55 | 321 / 328 | 0% | 0 | 0 / 178 |
heart | 97% | 429.58 | 833 / 861 | 0% | 0 | 0 / 0 |
liver | 95% | 514.90 | 215 / 226 | 0% | 0 | 0 / 406 |
muscle | 94% | 562.55 | 755 / 803 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 80 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
lymph node | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 29 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 45 |
ureter | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 1 |
GO_0032508 | Biological process | DNA duplex unwinding |
GO_0032680 | Biological process | regulation of tumor necrosis factor production |
GO_0006091 | Biological process | generation of precursor metabolites and energy |
GO_0006281 | Biological process | DNA repair |
GO_0034614 | Biological process | cellular response to reactive oxygen species |
GO_0043457 | Biological process | regulation of cellular respiration |
GO_0008340 | Biological process | determination of adult lifespan |
GO_0045184 | Biological process | establishment of protein localization |
GO_0006308 | Biological process | DNA catabolic process |
GO_0051607 | Biological process | defense response to virus |
GO_0061635 | Biological process | regulation of protein complex stability |
GO_0072711 | Biological process | cellular response to hydroxyurea |
GO_0160049 | Biological process | negative regulation of cGAS/STING signaling pathway |
GO_0001822 | Biological process | kidney development |
GO_0019217 | Biological process | regulation of fatty acid metabolic process |
GO_0046890 | Biological process | regulation of lipid biosynthetic process |
GO_0002637 | Biological process | regulation of immunoglobulin production |
GO_0050727 | Biological process | regulation of inflammatory response |
GO_0006110 | Biological process | regulation of glycolytic process |
GO_0006260 | Biological process | DNA replication |
GO_0002437 | Biological process | inflammatory response to antigenic stimulus |
GO_0032479 | Biological process | regulation of type I interferon production |
GO_0071480 | Biological process | cellular response to gamma radiation |
GO_1904161 | Biological process | DNA synthesis involved in UV-damage excision repair |
GO_0035458 | Biological process | cellular response to interferon-beta |
GO_0002383 | Biological process | immune response in brain or nervous system |
GO_0097281 | Biological process | immune complex formation |
GO_0045824 | Biological process | negative regulation of innate immune response |
GO_0002253 | Biological process | activation of immune response |
GO_0043277 | Biological process | apoptotic cell clearance |
GO_0060337 | Biological process | type I interferon-mediated signaling pathway |
GO_0003228 | Biological process | atrial cardiac muscle tissue development |
GO_0006259 | Biological process | DNA metabolic process |
GO_0050821 | Biological process | protein stabilization |
GO_0003015 | Biological process | heart process |
GO_0006304 | Biological process | DNA modification |
GO_0001568 | Biological process | blood vessel development |
GO_0002457 | Biological process | T cell antigen processing and presentation |
GO_0050863 | Biological process | regulation of T cell activation |
GO_0002281 | Biological process | macrophage activation involved in immune response |
GO_1905671 | Biological process | regulation of lysosome organization |
GO_0035781 | Biological process | CD86 biosynthetic process |
GO_0032197 | Biological process | retrotransposition |
GO_0006310 | Biological process | DNA recombination |
GO_0060339 | Biological process | negative regulation of type I interferon-mediated signaling pathway |
GO_0031571 | Biological process | mitotic G1 DNA damage checkpoint signaling |
GO_0006298 | Biological process | mismatch repair |
GO_0002320 | Biological process | lymphoid progenitor cell differentiation |
GO_0003007 | Biological process | heart morphogenesis |
GO_0043596 | Cellular component | nuclear replication fork |
GO_0032993 | Cellular component | protein-DNA complex |
GO_0008250 | Cellular component | oligosaccharyltransferase complex |
GO_0005789 | Cellular component | endoplasmic reticulum membrane |
GO_0005635 | Cellular component | nuclear envelope |
GO_0005829 | Cellular component | cytosol |
GO_0005737 | Cellular component | cytoplasm |
GO_0042803 | Molecular function | protein homodimerization activity |
GO_0008301 | Molecular function | DNA binding, bending |
GO_0032407 | Molecular function | MutSalpha complex binding |
GO_0003697 | Molecular function | single-stranded DNA binding |
GO_0003690 | Molecular function | double-stranded DNA binding |
GO_0008296 | Molecular function | 3'-5'-DNA exonuclease activity |
GO_0032405 | Molecular function | MutLalpha complex binding |
GO_0000287 | Molecular function | magnesium ion binding |
GO_0008408 | Molecular function | 3'-5' exonuclease activity |
GO_0050699 | Molecular function | WW domain binding |
GO_0008311 | Molecular function | double-stranded DNA 3'-5' DNA exonuclease activity |
GO_0046872 | Molecular function | metal ion binding |
GO_0032558 | Molecular function | adenyl deoxyribonucleotide binding |
Gene name | TREX1 |
Protein name | Three-prime repair exonuclease 1 (EC 3.1.11.2) (3'-5' exonuclease TREX1) (Deoxyribonuclease III) (DNase III) exodeoxyribonuclease III (EC 3.1.11.2) Alternative protein TREX1 |
Synonyms | hCG_1997849 |
Description | FUNCTION: Major cellular 3'-to-5' DNA exonuclease which digests single-stranded DNA (ssDNA) and double-stranded DNA (dsDNA) with mismatched 3' termini . Prevents cell-intrinsic initiation of autoimmunity . Acts by metabolizing DNA fragments from endogenous retroelements, including L1, LTR and SINE elements . Plays a key role in degradation of DNA fragments at cytosolic micronuclei arising from genome instability: its association with the endoplasmic reticulum membrane directs TREX1 to ruptured micronuclei, leading to micronuclear DNA degradation . Micronuclear DNA degradation is required to limit CGAS activation and subsequent inflammation . Unless degraded, these DNA fragments accumulate in the cytosol and activate the cGAS-STING innate immune signaling, leading to the production of type I interferon . Prevents chronic ATM-dependent checkpoint activation, by processing ssDNA polynucleotide species arising from the processing of aberrant DNA replication intermediates . Inefficiently degrades oxidized DNA, such as that generated upon antimicrobial reactive oxygen production or upon absorption of UV light . During GZMA-mediated cell death, contributes to DNA damage in concert with NME1 . NME1 nicks one strand of DNA and TREX1 removes bases from the free 3' end to enhance DNA damage and prevent DNA end reannealing and rapid repair . . |
Accessions | ENST00000444177.1 [Q9NSU2-2] ENST00000492235.2 ENST00000635452.2 Q6IAN5 ENST00000625293.3 [Q9NSU2-3] Q9NSU2 ENST00000456089.1 L8E8B0 ENST00000433541.1 Q5TZT0 C9J052 |