Insufficient scRNA-seq data for expression of PANK4 at single-cell level.
Insufficient scRNA-seq data for expression of PANK4 at tissue level.
Tissue | GTEx Coverage | GTEx Average TPM | GTEx Number of samples | TCGA Coverage | TCGA Average TPM | TCGA Number of samples |
---|---|---|---|---|---|---|
esophagus | 100% | 1679.87 | 1445 / 1445 | 100% | 12.43 | 183 / 183 |
skin | 100% | 1638.78 | 1808 / 1809 | 100% | 22.43 | 472 / 472 |
prostate | 100% | 1628.00 | 245 / 245 | 100% | 15.33 | 501 / 502 |
uterus | 100% | 1730.09 | 170 / 170 | 100% | 17.67 | 458 / 459 |
lung | 100% | 1806.37 | 577 / 578 | 100% | 14.46 | 1153 / 1155 |
bladder | 100% | 1799.43 | 21 / 21 | 100% | 15.61 | 502 / 504 |
kidney | 100% | 1395.94 | 89 / 89 | 100% | 12.09 | 897 / 901 |
thymus | 100% | 1867.37 | 653 / 653 | 100% | 18.49 | 602 / 605 |
pancreas | 100% | 720.86 | 328 / 328 | 99% | 13.98 | 177 / 178 |
stomach | 100% | 1315.66 | 359 / 359 | 99% | 12.21 | 284 / 286 |
breast | 100% | 1499.75 | 459 / 459 | 99% | 13.80 | 1110 / 1118 |
intestine | 100% | 1874.09 | 966 / 966 | 99% | 13.06 | 523 / 527 |
adrenal gland | 100% | 1518.78 | 258 / 258 | 99% | 15.41 | 228 / 230 |
ovary | 100% | 1327.45 | 180 / 180 | 99% | 12.26 | 426 / 430 |
brain | 99% | 1494.21 | 2617 / 2642 | 100% | 19.68 | 705 / 705 |
liver | 100% | 979.67 | 226 / 226 | 97% | 8.69 | 392 / 406 |
adipose | 100% | 1448.44 | 1204 / 1204 | 0% | 0 | 0 / 0 |
eye | 0% | 0 | 0 / 0 | 100% | 20.38 | 80 / 80 |
lymph node | 0% | 0 | 0 / 0 | 100% | 16.26 | 29 / 29 |
muscle | 100% | 2444.10 | 803 / 803 | 0% | 0 | 0 / 0 |
spleen | 100% | 2091.60 | 241 / 241 | 0% | 0 | 0 / 0 |
tonsil | 0% | 0 | 0 / 0 | 100% | 17.79 | 45 / 45 |
ureter | 0% | 0 | 0 / 0 | 100% | 7.97 | 1 / 1 |
blood vessel | 100% | 1398.15 | 1332 / 1335 | 0% | 0 | 0 / 0 |
heart | 99% | 1870.35 | 850 / 861 | 0% | 0 | 0 / 0 |
peripheral blood | 99% | 1481.11 | 916 / 929 | 0% | 0 | 0 / 0 |
abdomen | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
bone marrow | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
diaphragm | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
gingiva | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasal cavity | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nasopharynx | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
nose | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
placenta | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
spinal column | 0% | 0 | 0 / 0 | 0% | 0 | 0 / 0 |
GO_0015939 | Biological process | pantothenate metabolic process |
GO_0015937 | Biological process | coenzyme A biosynthetic process |
GO_0005829 | Cellular component | cytosol |
GO_0005634 | Cellular component | nucleus |
GO_0016791 | Molecular function | phosphatase activity |
GO_0004594 | Molecular function | pantothenate kinase activity |
GO_0046872 | Molecular function | metal ion binding |
GO_0005524 | Molecular function | ATP binding |
Gene name | PANK4 |
Protein name | Pantothenate kinase 4 putative variant (Pantothenate kinase 4, isoform CRA_b) 4'-phosphopantetheine phosphatase (Inactive pantothenic acid kinase 4) 4'-phosphopantetheine phosphatase (EC 3.1.3.-) (Inactive pantothenic acid kinase 4) (hPanK4) 4'-phosphopantetheine phosphatase (EC 3.1.3.-) 4'-phosphopantetheine phosphatase (Pantothenate kinase 4 (inactive)) |
Synonyms | hCG_24601 |
Description | FUNCTION: Phosphatase which shows a preference for 4'-phosphopantetheine and its oxidatively damaged forms (sulfonate or S-sulfonate), providing strong indirect evidence that the phosphatase activity pre-empts damage in the coenzyme A (CoA) pathway . Hydrolyzing excess 4'-phosphopantetheine could constitute a directed overflow mechanism to prevent its oxidation to the S-sulfonate, sulfonate, or other forms . Hydrolyzing 4'-phosphopantetheine sulfonate or S-sulfonate would forestall their conversion to inactive forms of CoA and acyl carrier protein . May play a role in the physiological regulation of CoA intracellular levels (Probable). . FUNCTION: Phosphatase which shows a preference for 4'-phosphopantetheine and its oxidatively damaged forms (sulfonate or S-sulfonate), providing strong indirect evidence that the phosphatase activity pre-empts damage in the coenzyme A (CoA) pathway. Hydrolyzing excess 4'-phosphopantetheine could constitute a directed overflow mechanism to prevent its oxidation to the S-sulfonate, sulfonate, or other forms. Hydrolyzing 4'-phosphopantetheine sulfonate or S-sulfonate would forestall their conversion to inactive forms of CoA and acyl carrier protein. May play a role in the physiological regulation of CoA intracellular levels. . |
Accessions | H0Y9E4 ENST00000435556.8 ENST00000505228.5 ENST00000502512.1 ENST00000625442.1 ENST00000468002.2 ENST00000626423.2 D6RJF3 ENST00000628687.1 Q9NVE7 ENST00000378466.9 H0YA31 ENST00000486396.1 ENST00000630982.2 ENST00000615291.3 H0YA26 E9PHT6 ENST00000502770.2 ENST00000628406.1 ENST00000627774.1 H0YA02 Q7RTX2 |